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종합병원 입원 환자의 간호대학생 실습허용에 대한 견해 및 인식
김영진 ( Kim Youngjin ),김지선 ( Kim Jeesun ),김혜림 ( Kim Hyerim ),박수비 ( Park Subi ),소샛별 ( So Saetbyul ),정보성 ( Jung Bosung ),조은애 ( Choh Eunae ),이선영 ( Lee Seonyoung ),김정은 ( Kim Jeongeun ) 서울대학교 간호과학연구소 2017 간호학의 지평 Vol.14 No.1
Purpose: The aim of this study was to explore the patients` perspectives on nursing students` clinical practices in the wards, and to investigate their willingness for allowing students to practice on them. Methods: This was a descriptive study. 116 inpatients were recruited from the S University Hospital. A 60-item questionnaire was applied to collect the data. The participants were 19 years and older with sound judgement, and were not in special or intensive care units. Data analysis was done in SPSS/WIN 22.0 using descriptive statistics, Fishers exact test, and the ANOVA test. the participant answered to questionnaire from April 29th 2016 to May 10th. Results: 40 participants (34.5%) stated they would allow students` practice, while 72 (61.2%) said they would allow only under staff supervision. 5 participants (4.3%) stated they would not allow whatsoever. The 3 most allowed were emotional support, oral care, and vital signs measurement while the 3 least allowed were gastric feeding, intravenous catheterization, and urinary catheterization. Conclusion: Patients were more inclined to allow students to practice on them when a member of the medical team was present. A fair number of participants said they would be more inclined to allow students` practice if they felt the student was competent; hence, reinforcing simulation sessions is vital in enhancing students` competency and ultimately practice allowance.
Oh, Soojung,Lee, Mak-Soon,Jung, Sunyoon,Kim, Seunghae,Park, Hyeyoung,Park, Seonyoung,Kim, Seog-Young,Kim, Chong-Tai,Jo, Young-Hee,Kim, In-Hwan,Kim, Yangha Elsevier 2017 Journal of Functional Foods Vol.29 No.-
<P><B>Abstract</B></P> <P>Mitochondrial dysfunction and dyslipidemia are associated with obesity-linked metabolic disorders. The aim of this study was to investigate the effects of ginger extract on muscle mitochondrial biogenesis and high-density lipoprotein-cholesterol (HDL-C) metabolism in high-fat diet-fed rats. Supplementation with ginger extract reduced final body weight and epididymal adipose tissue mass without affecting energy intake (<I>p</I> <0.05). Ginger extract increased mitochondrial size and mitochondrial DNA (mtDNA) content as well as key genes expression related to mitochondrial biogenesis, including peroxisome proliferator-activated receptor γ coactivator-1α (PGC-1α), nuclear respiratory factor-1 (NRF-1) and transcription factor A (Tfam) in skeletal muscle (<I>p</I> <0.05). In addition, ginger extract elevated serum HDL-C along with up-regulating ATP-binding cassette transporter A1 (ABCA1), apolipoprotein A-1 (ApoA-1) and lecithin-cholesterol acyltransferase (LCAT) mRNA in liver (<I>p</I> <0.05). These results suggest that ginger extract may have a beneficial effect against obesity-related metabolic disorders with elevating muscle mitochondrial biogenesis and serum HDL-C level.</P> <P><B>Highlights</B></P> <P> <UL> <LI> Effect of ginger extract was investigated in high-fat diet-fed rats. </LI> <LI> Treatment with ginger extract increased muscle mitochondrial biogenesis. </LI> <LI> Treatment with ginger extract elevated serum high-density lipoprotein-cholesterol level. </LI> <LI> Taken together, ginger extract might have beneficial effect against obesity-related metabolic disorders. </LI> </UL> </P>
Circulating Tumor Cell Microseparator Based on Lateral Magnetophoresis and Immunomagnetic Nanobeads
Kim, Seonyoung,Han, Song-I,Park, Min-Jae,Jeon, Chang-Wan,Joo, Young-Don,Choi, In-Hak,Han, Ki-Ho American Chemical Society 2013 ANALYTICAL CHEMISTRY - Vol.85 No.5
<P>This paper presents a circulating tumor cell (CTC) microseparator for isolation of CTCs from human peripheral blood using immunomagnetic nanobeads with bound antiepithelial cell adhesive molecule (EpCAM) antibodies that specifically bind to epithelial cancer cells. The isolation is performed through lateral magnetophoresis, which is induced by high-gradient magnetic separation technology, involving a ferromagnetic wire array inlaid in the bottom substrate of a microchannel. Experimental results showed that the CTC microseparator isolates about 90% of spiked CTCs in human peripheral blood at a flow rate of up to 5 mL/h and purifies to approximately 97%. The overall isolation procedure was completed within 15 min for 200 μL of peripheral blood. CTCs from peripheral blood of patients with breast and lung cancers were isolated with the CTC microseparator, and the results were compared with those of healthy donors. Using a fluorescence-based viability assay, the viability of CTCs isolated from peripheral blood of patients with cancer was observed. In addition, the usefulness of the CTC microseparator for subsequent genetic assay was confirmed by reverse-transcriptase polymerase chain reaction (RT-PCR) amplification of cancer-specific genes using CTCs isolated from patients with cancer.</P><P><B>Graphic Abstract</B> <IMG SRC='http://pubs.acs.org/appl/literatum/publisher/achs/journals/content/ancham/2013/ancham.2013.85.issue-5/ac303284u/production/images/medium/ac-2012-03284u_0009.gif'></P><P><A href='http://pubs.acs.org/doi/suppl/10.1021/ac303284u'>ACS Electronic Supporting Info</A></P>
( Seonyoung Kim ),( Seok-seong Kang ),( Soo-im Choi ),( Gun-hee Kim ),( Jee-young Imm ) 한국미생물 · 생명공학회 2019 Journal of microbiology and biotechnology Vol.29 No.1
Ecklonia cava, an edible marine brown alga (Laminariaceae), is a rich source of bioactive compounds such as fucoidan and phlorotannins. Ecklonia cava extract (ECE) was prepared using 70% ethanol extraction and ECE contained 67% and 10.6% of total phlorotannins and dieckol, respectively. ECE treatment significantly inhibited receptor activator of nuclear factor-κB ligand (RANKL)-induced osteoclast differentiation of RAW 264.7 cells and pit formation in bone resorption assay (p <0.05). Moreover, it suppressed RANKL-induced NF-κB and mitogen-activated protein kinase signaling in a dose dependent manner. Downregulated osteoclast-specific gene (tartrate-resistant acid phosphatase, cathepsin K, and matrix metalloproteinase-9) expression and osteoclast proliferative transcriptional factors (nuclear factor of activated T cells-1 and c-fos) confirmed ECE-mediated suppression of osteoclastogenesis. ECE treatment (100 μg/ml) increased heme oxygenase-1 expression by 2.5-fold and decreased intercellular reactive oxygen species production during osteoclastogenesis. The effective inhibition of RANKL-stimulated osteoclast differentiation and oxidative stress by ECE suggest that ECE has therapeutic potential in alleviating osteoclast-associated disorders.