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Pyun, So Young,Kim, Byung-Jo The Korean Society of Clinical Neurophysiology 2017 Annals of Clinical Neurophysiology Vol.19 No.2
Paraproteinemia is caused by a proliferation of monoclonal plasma cells or B lymphocytes. Approximately 10% of idiopathic neuropathies are associated with paraproteinemia, where a certain paraprotein acts like an antibody targeted at constituents of myelin or axolemma in peripheral nerves. The relationship between paraproteinemia and peripheral neuropathy remains unclear despite this being of interest for a long time. Neurologists frequently find paraproteinemia during laboratory examinations of patients presenting with peripheral neuropathy, especially in the elderly. The possibility of a relationship with paraproteinemia should be considered in cases without an explainable cause. We review the causal association between paraproteinemia and neuropathy as well as clinical, laboratory, and electrophysiologic features, and the treatment options for paraproteinemic neuropathy.
Gender Differences in Korean Patients with Obstructive Sleep Apnea
Pyun So Young,최수정,조현진,황윤하,Cho Jae Wook,Joo Eun Yeon 대한수면학회 2020 sleep medicine research Vol.11 No.2
Background and ObjectiveaaOur objective in this study was to investigate gender-specific differences in demographic factors, clinical presenting symptoms, medical comorbidities, and sleeprelated parameters in patients newly diagnosed with obstructive sleep apnea (OSA). MethodsaaWe enrolled patients who were more than 30 years old, had undergone overnight polysomnography, and had filled out the questionnaires, including demographics, medical comorbidities, and sleep-related symptoms. ResultsaaFrom December 2014 to August 2017, 1224 patients (female, n = 277, 22.6%) were newly diagnosed with OSA. Female patients were significantly older than males (59.62 years vs. 52.75 years, p < 0.001). Alcohol consumption and current smoking were more common in males (p < 0.001). Non-specific symptoms of OSA, such as insomnia-related symptoms, loss of energy, and subjective poor sleep, were observed more frequently in females. Females reported more depressive moods (Beck Depression Inventory-II 15.58) than males did (12.17). Males complained about OSA-specific symptoms, such as snoring, witnessed apnea, and daytime sleepiness, more frequently than females did. The mean apnea-hypopnea index (AHI) was much lower in females (26.25 /h vs. 33.36 /h), but AHI during rapid eye movement sleep was similar for the two groups (33.55 /h vs. 32.76 /h). Sleep latency was longer (15.45 min vs. 10.11 min) and spontaneous arousal index (4.48 /h vs. 3.48 /h) was more frequent in females. ConclusionsaaFemales have a different OSA phenotype from males in terms of demographics, clinical manifestations, and sleep parameters of polysomnography. Thus, clinicians should comprehend the different clinical phenotype of female OSA and detect earlier unrecognized OSA in females.
Chemistry of Carbonate-Sulfur Flux
Q. Won Choi,Choi Han,Chang So-Young,Pyun Chong-Hong,Kim Chang-Hong Korean Chemical Society 1994 Bulletin of the Korean Chemical Society Vol.15 No.12
Reactions of alkaline metal carbonates with sulfur are investigated in detail. The evolution of CO and a trace of $SO_2$ were observed in the course of reaction with major component of polysulfides. Some evidences that the reaction proceeds with breaking of terminal sulfur-sulfur bond in the sulfur polymer, and forming CO, $SO_2$ and polysulfide are presented. Polysulfides have the role of keeping free sulfur and allow it to react with other chemicals to rather high temperatures.plexes, whereas the binuclear and mononuclear complexes of Mn$^{2+}$ and Co$^{2+}$</TE. showed similar reactivities. The nature of the ligands bound to copper did not influence the reactivity of the binuclear copper complexes so long as copper ions were held in close proximity. A metal-iodosylbenzene complex, such as suggested previously for Lewis acidic metal complex-catalyzed epoxidat.n by iodosylbenzene, is proposed as the active epoxidizing species. Some mechanistic aspects are discussed as well.
Normal data on axonal excitability in Koreans
Lee, Ju Young,Yu, Jin Hyeok,Pyun, So Young,Ryu, Sanghyo,Bae, Jong Seok The Korean Society of Clinical Neurophysiology 2017 Annals of Clinical Neurophysiology Vol.19 No.1
Background: Automated nerve excitability testing is used to assess various peripheral neuropathies and motor neuron diseases. Comparing these excitability parameters with normal data provides information regarding the axonal excitability properties and ion biophysics in diseased axons. This study measured and compared normal values of axonal excitability parameters in both the distal motor and sensory axons of normal Koreans. Methods: The axonal excitability properties of 50 distal median motor axons and 30 distal median sensory axons were measured. An automated nerve excitability test was performed using the QTRACW threshold-tracking software (Institute of Neurology, University College London, London, UK) with the TRONDF multiple excitability recording protocol. Each parameter of stimulus-response curves, threshold electrotonus, current-voltage relationship, and recovery cycle was measured and calculated. Results: Our Korean normal data on axonal excitability showed ranges of values and characteristics similar to previous reports from other countries. We also reaffirmed that there exist characteristic differences in excitability properties between motor and sensory axons: compared to motor axons, sensory axons showed an increased strength-duration time constant, more prominent changes in threshold to hyperpolarizing threshold electrotonus (TE) and less prominent changes in threshold to depolarizing TE, and more prominent refractoriness and less prominent subexcitability and superexcitability. Conclusions: We report normal data on axonal excitability in Koreans. These data can be used to compare various pathological conditions in peripheral nerve axons such as peripheral neuropathies and motor neuron disease.
변소영,김병조,Pyun, So Young,Kim, Byung-Jo 대한임상신경생리학회 2015 Annals of Clinical Neurophysiology Vol.17 No.2
The paraproteinemia is a disorder in which a single clone of plasma cells (monoclonal gammopathy) is responsible for the proliferation of monoclonal proteins (M-proteins). Approximately 10% of patients with idiopathic peripheral neuropathy have monoclonal gammopathy. Some M-proteins have the properties of an antibody to the components of peripheral nerve myelin, but the pathophysiological relationship between the neuropathy and the M-protein is often obscure. The relationship between peripheral neuropathy and monoclonal gammopathy requires the appropriate neurological and hematological investigations for precise diagnosis and treatment. In this review, we provide an update on the causal associations between peripheral neuropathy and monoclonal gammopathy as well as characteristics of clinical and electrophysiologic features.