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Domain Wall Pinning by a Stray Field from NiFe on a Co/Ni Nanowire
R. Hiramatsu,T. Koyama,H. Hata,T. Ono,D. Chiba,S. Fukami,N. Ishiwata 한국물리학회 2013 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.63 No.3
We have investigated the influence of a stray field on a magnetic domain wall (DW). Thedepinning fields of a DW in a perpendicularly magnetized Co/Ni nanowire with partly-stackedNi81Fe19/SiO2 islands on it was measured. The depinning field for the wire with Ni81Fe19/SiO2was found to be higher than that for the wire without the stack, indicating that the stray field fromthe Ni81Fe19/SiO2 stack clearly served as an artificial pinning site for the DW. The magnetic forcemicroscopy observation evidenced that the DW was pinned near the Ni81Fe19 stack.
Toyoshima, R.,Hiramatsu, N.,Yoshida, M.,Amemiya, K.,Mase, K.,Mun, B.,Kondoh, H. Royal Society of Chemistry 2017 Chemical communications Vol.53 No.94
<P>Catalytic CO oxidation over Pd(111) and Pd70Au30(111) surfaces was investigated by in situ spectroscopic observations to understand the alloying effect. The reaction behaviour on Pd70Au30(111) is greatly different from that on Pd(111). Pd monomer and dimer ensembles can act as active centers, whereas triangular-shaped trimers and larger ensembles are inactive.</P>
Alkam, T.,Mamiya, T.,Kimura, N.,Yoshida, A.,Kihara, D.,Tsunoda, Y.,Aoyama, Y.,Hiramatsu, M.,Kim, H. C.,Nabeshima, T. Springer Science + Business Media 2017 Psychophamacology Vol.234 No.12
<P>Increased risk of attention-deficit/hyperactivity disorder (AD/HD) is partly associated with the early developmental exposure to nicotine in tobacco smoke. Emerging reports link tobacco smoke exposure or prenatal nicotine exposure (PNE) with AD/HD-like behaviors in rodent models. We have previously reported that PNE induces cognitive behavioral deficits in offspring and decreases the contents of dopamine (DA) and its turnover in the prefrontal cortex (PFC) of offspring It is well known that the dysfunction of DAergic system in the brain is one of the core factors in the pathophysiology of AD/HD. Therefore, we examined whether the effects of PNE on the DAergic system underlie the AD/HD-related behavioral changes in mouse offspring. PNE reduced the release of DA in the medial PFC (mPFC) in mouse offspring. PNE reduced the number of tyrosine hydroxylase (TH)-positive varicosities in the mPFC and in the core as well as the shell of nucleus accumbens, but not in the striatum. PNE also induced behavioral deficits in cliff avoidance, object-based attention, and sensorimotor gating in offspring. These behavioral deficits were attenuated by acute treatment with atomoxetine (3 mg/kg, s.c.) or partially attenuated by acute treatment with MPH (1 mg/kg, s.c.). Taken together, our findings support the notion that PNE induces neurobehavioral abnormalities in mouse offspring by disrupting the DAergic system and improve our understanding about the incidence of AD/HD in children whose mothers were exposed to nicotine during their pregnancy.</P>