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Kim, Joonyup,Yang, Ronghui,Chang, Caren,Park, Younghoon,Tucker, Mark L Oxford University Press 2018 Journal of experimental botany Vol.69 No.12
<▼1><P>A root-knot nematode gene (<I>MiIDL1</I>) produces a peptide that mimics the Arabidopsis IDA signaling peptide that regulates cell separation events including abscission and lateral root emergence.</P></▼1><▼2><P><B>Abstract</B></P><P>INFLORESCENCE DEFICIENT IN ABSCISSION (IDA) is a signaling peptide that regulates cell separation in Arabidopsis including floral organ abscission and lateral root emergence. IDA is highly conserved in dicotyledonous flowering plant genomes. <I>IDA-like</I> sequences were also found in the genomic sequences of root-knot nematodes, <I>Meloidogyne</I> spp., which are globally deleterious pathogens of agriculturally important plants, but the role of these genes is unknown. Exogenous treatment of the Arabidopsis <I>ida</I> mutant with synthetic peptide identical to the <I>M. incognita</I> IDA-like 1 (MiIDL1) protein sequence minus its N-terminal signal peptide recovered both the abscission and root architecture defects. Constitutive expression of the full-length <I>MiIDL1</I> open reading frame in the <I>ida</I> mutant substantially recovered the delayed floral organ abscission phenotype whereas transformants expressing a construct missing the MiIDL1 signal peptide retained the delayed abscission phenotype. Importantly, wild-type Arabidopsis plants harboring an <I>MiIDL1</I>-RNAi construct and infected with nematodes had approximately 40% fewer galls per root than control plants. Thus, the <I>MiIDL1</I> gene produces a functional IDA mimic that appears to play a role in successful gall development on Arabidopsis roots.</P></▼2>