http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Preparation and properties of CNTs-Cu hybrids/epoxy superhydrophobic and anticorrosive coatings
Zhongbo Zhu,Shumei Kang,Hua Chen,Qingping Zhao,Zhaokang Huo,Pengyu Li,Qidong Cao,Chenshuo Lu 한국공업화학회 2023 Journal of Industrial and Engineering Chemistry Vol.119 No.-
In this work, hydrophobized copper powders combined siloxane-modified carbon nanotubes(SHMWCNTs) to obtain intertwined hybrids of carbon nanotubes and copper (CNTs-Cu hybrids), andsuperhydrophobic CNTs-Cu hybrids/epoxy coatings were prepared on the surface of AH36 carbon steelby drop coating. The surface morphology and wettability of coatings were changed by adjusting the ratiosof SHMWCNTs to hydrophobized copper powders. The obtained coating can show a water contact angleof 167.2, a water sliding angle of 2.9, a plant oil contact angle of 134.3, and self-cleaning properties. Thecoating maintains superhydrophobicity after 300 tape-peeling times, exhibiting excellent mechanicaldurability. The fouled area percentages of chlorella on the coating and a pure epoxy resin were 0.23(±0.07)% and 12.18(±1.22)%, respectively, displaying anti-biofouling resistances. The coating has excellentcorrosion resistance, and its corrosion current density reached 9.55 10-8 A/cm2.
Chen Kun,Chen Xi,Lang Chuandong,Yuan Xingshi,Huang Junming,Li Zhi,Xu Mingyou,Wu Kerong,Zhou Chenhe,Li Qidong,Zhu Chen,Liu Lianxin,Shang Xifu 생화학분자생물학회 2023 Experimental and molecular medicine Vol.55 No.-
The identification of key regulatory factors that control osteoclastogenesis is important. Accumulating evidence indicates that circular RNAs (circRNAs) are discrete functional entities. However, the complexities of circRNA expression as well as the extent of their regulatory functions during osteoclastogenesis have yet to be revealed. Here, based on circular RNA sequencing data, we identified a circular RNA, circFam190a, as a critical regulator of osteoclast differentiation and function. During osteoclastogenesis, circFam190a is significantly upregulated. In vitro, circFam190a enhanced osteoclast formation and function. In vivo, overexpression of circFam190a induced significant bone loss, while knockdown of circFam190a prevented pathological bone loss in an ovariectomized (OVX) mouse osteoporosis model. Mechanistically, our data suggest that circFam90a enhances the binding of AKT1 and HSP90β, promoting AKT1 stability. Altogether, our findings highlight the critical role of circFam190a as a positive regulator of osteoclastogenesis, and targeting circFam190a might be a promising therapeutic strategy for treating pathological bone loss.