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Lim, Suk-Kyung,Tanimoto, Koichi,Tomita, Haruyoshi,Ike, Yasuyoshi American Society for Microbiology 2006 Applied and environmental microbiology Vol.72 No.10
<B>ABSTRACT</B><P>The drug resistances and plasmid contents of a total of 85 vancomycin-resistant enterococcus (VRE) strains that had been isolated in Korea were examined. Fifty-four of the strains originated from samples of chicken feces, and 31 were isolated from hospital patients in Korea. <I>Enterococcus faecalis</I> KV1 and KV2, which had been isolated from a patient and a sample of chicken feces, respectively, were found to carry the plasmids pSL1 and pSL2, respectively. The plasmids transferred resistances to vancomycin, gentamicin, kanamycin, streptomycin, and erythromycin to <I>E. faecalis</I> strains at a high frequency of about 10<SUP>−3</SUP> per donor cell during 4 hours of broth mating. <I>E. faecalis</I> strains containing each of the pSL plasmids formed clumps after 2 hours of incubation in broth containing <I>E. faecalis</I> FA2-2 culture filtrate (i.e., the <I>E. faecalis</I> sex pheromone), and the plasmid subsequently transferred to the recipient strain in a 10-min short mating in broth, indicating that the plasmids are responsive to <I>E. faecalis</I> pheromones. The pSL plasmids did not respond to any of synthetic pheromones for the previously characterized plasmids. The pheromone specific for pSL plasmids has been designated cSL1. Southern hybridization analysis showed that specific FspI fragments from each of the pSL plasmids hybridized with the aggregation substance gene (<I>asa1</I>) of the pheromone-responsive plasmid pAD1, indicating that the plasmids had a gene homologous to <I>asa1</I>. The restriction maps of the plasmids were identical, and the size of the plasmids was estimated to be 128.1 kb. The plasmids carried five drug resistance determinants for <I>vanA</I>, <I>ermB</I>, <I>aph(3</I>′<I>)</I>, <I>aph(6</I>′<I>)</I>, and <I>aac(6</I>′<I>)</I>/<I>aph(2</I>′<I>)</I>, which encode resistance to vancomycin, erythromycin, kanamycin, streptomycin, and gentamicin/kanamycin, respectively. Nucleotide sequence analyses of the drug resistance determinants and their flanking regions are described in this report. The results described provide evidence for the exchange of genetic information between human and animal (chicken) VRE reservoirs and suggest the potential for horizontal transmission of multiple drug resistance, including vancomycin resistance, between farm animals and humans via a pheromone-responsive conjugative plasmid.</P>
Jung, Woo Kyung,Hong, Soon Keun,Lim, Ji Youn,Lim, Suk Kyung,Kwon, Nam Hoon,Kim, Jun Man,Koo, Hye Cheong,Kim, So Hyun,Seo, Keun Seok,Ike, Yasuyoshi,Tanimoto, Koichi,Park, Yong Ho Oxford University Press 2006 FEMS microbiology letters Vol.260 No.2
<P>Vancomycin resistant enterococci (VRE) isolates from humans (23 isolates) and poultry (20 isolates) were characterized by antibiotic susceptibility, vancomycin resistance transferability, pulsed-field gel electrophoresis (PFGE), and structural analysis of Tn1546-like elements. VRE isolates from humans and poultry showed different resistance patterns, transferability, and transfer rate. In addition to these phenotypic differences between humans and poultry VRE, PFGE and the structure of Tn1546-like elements were also distinct. Most poultry isolates (16/20) were identical to the prototype vanA transposon, Tn1546, while most human isolates (21/23) had multiple integrations of insertion sequence. The transmission of VRE and vancomycin resistance determinant between humans and poultry could not be demonstrated in this study.</P>