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이정일(Jung Il Lee),박장환(Jang Hwan Park),김석동(Sok Dong Kim),안병옥(Byeong Ok Ahn),이승택(Seung Tack Lee) 한국약용작물학회 1993 한국약용작물학회지 Vol.1 No.1
This study was conducted to select thin-shelled and high-yielding lines in job`s-tears. Two breeding lines of Suwon 3 and Suwon 6 were selected from the local collections. These two lines were tested and investigated on their characteristics under the field condition. The heading date of Suwon 3 and Suwon 6 was later one or two days, but the maturity date was one or two days earlier than that of check variety Kim-jejong, respectively. The number of grains per hill of Suwon 3, Suwon 6 was 50%, 49% greater and the milling rate was 3.8%, 5.6% higher than that of check variety, respectively. Althought 1000 grain weight of Suwon 3 and Suwon 6 was 20g lighter and the rate of ripeness was 6%, 12% lower, the raw grain yield was 22%, 20% higher than that of check variety, respectively. The thickness of seed coat of Suwon 3 and Suwon 6 was thiner and the hardness of seed coat was lower than that of check variety, therefore the milling time was decreased 12%, 7% compare to check variety, respectively. The crude protein contents of Suwon 3 and Suwon 6 was slightly higher and the amino acid composition of Suwon 6 was similar to Kimjejong, but Suwon 3 was lower than that of check variety.
Kim, Hyo Jung,Hong, Sung Hyun,Kim, You Wang,Lee, Il Hwan,Jun, Ji Hyung,Phee, Bong-Kwan,Rupak, Timilsina,Jeong, Hana,Lee, Yeonmi,Hong, Byoung Seok,Nam, Hong Gil,Woo, Hye Ryun,Lim, Pyung Ok Oxford University Press 2014 Journal of experimental botany Vol.65 No.14
<P>Leaf senescence is a finely tuned and genetically programmed degeneration process, which is critical to maximize plant fitness by remobilizing nutrients from senescing leaves to newly developing organs. Leaf senescence is a complex process that is driven by extensive reprogramming of global gene expression in a highly coordinated manner. Understanding how gene regulatory networks involved in controlling leaf senescence are organized and operated is essential to decipher the mechanisms of leaf senescence. It was previously reported that the trifurcate feed-forward pathway involving <I>EIN2</I>, <I>ORE1</I>, and <I>miR164</I> in <I>Arabidopsis</I> regulates age-dependent leaf senescence and cell death. Here, new components of this pathway have been identified, which enhances knowledge of the gene regulatory networks governing leaf senescence. Comparative gene expression analysis revealed six senescence-associated NAC transcription factors (TFs) (ANAC019, AtNAP, ANAC047, ANAC055, ORS1, and ORE1) as candidate downstream components of ETHYLENE-INSENSITIVE2 (EIN2). EIN3, a downstream signalling molecule of EIN2, directly bound the <I>ORE1</I> and <I>AtNAP</I> promoters and induced their transcription. This suggests that EIN3 positively regulates leaf senescence by activating <I>ORE1</I> and <I>AtNAP</I>, previously reported as key regulators of leaf senescence. Genetic and gene expression analyses in the <I>ore1 atnap</I> double mutant revealed that ORE1 and AtNAP act in distinct and overlapping signalling pathways. Transient transactivation assays further demonstrated that ORE1 and AtNAP could activate common as well as differential NAC TF targets. Collectively, the data provide insight into an EIN2-mediated senescence signalling pathway that coordinates global gene expression during leaf senescence via a gene regulatory network involving EIN3 and senescence-associated NAC TFs.</P>
Kim, Hyo Jung,Park, Ji-Hwan,Kim, Jingil,Kim, Jung Ju,Hong, Sunghyun,Kim, Jeongsik,Kim, Jin Hee,Woo, Hye Ryun,Hyeon, Changbong,Lim, Pyung Ok,Nam, Hong Gil,Hwang, Daehee National Academy of Sciences 2018 PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF Vol.115 No.21
<▼1><P><B>Significance</B></P><P>Leaf senescence is regulated in a complex manner, involving time-dependent interactions with developmental and environmental signals. Genetic screens have identified key regulators of senescence, particularly late-stage senescence regulators. Recently, time-course gene-expression and network analyses, mostly analyses of static networks, have predicted many senescence regulators. However, senescence is defined by time-evolving networks, involving the temporal transition of interactions among senescence regulators. Here, we present time-evolving networks of NAM/ATAF/CUC (NAC) transcription factors, central regulators of leaf senescence in <I>Arabidopsis</I>, via time-course gene-expression analysis of NACs in their mutants. These time-evolving networks revealed a unique regulatory module of NACs that controls the timely induction of senescence-promoting processes at a presenescent stage of leaf aging.</P></▼1><▼2><P>Senescence is controlled by time-evolving networks that describe the temporal transition of interactions among senescence regulators. Here, we present time-evolving networks for NAM/ATAF/CUC (NAC) transcription factors in <I>Arabidopsis</I> during leaf aging. The most evident characteristic of these time-dependent networks was a shift from positive to negative regulation among NACs at a presenescent stage. ANAC017, ANAC082, and ANAC090, referred to as a “NAC troika,” govern the positive-to-negative regulatory shift. Knockout of the NAC troika accelerated senescence and the induction of other <I>NAC</I>s, whereas overexpression of the NAC troika had the opposite effects. Transcriptome and molecular analyses revealed shared suppression of senescence-promoting processes by the NAC troika, including salicylic acid (SA) and reactive oxygen species (ROS) responses, but with predominant regulation of SA and ROS responses by ANAC090 and ANAC017, respectively. Our time-evolving networks provide a unique regulatory module of presenescent repressors that direct the timely induction of senescence-promoting processes at the presenescent stage of leaf aging.</P></▼2>
Kim, Kyung Hyun,Cho, Ga Youn,Chun, Byung Hee,Weckx, Stefan,Moon, Ji Young,Yeo, Soo-Hwan,Jeon, Che Ok Elsevier 2018 Systematic and applied microbiology Vol.41 No.4
<P><B>Abstract</B></P> <P>Twelve <I>Acetobacter pasteurianus</I>-related strains with publicly available genomes in GenBank shared high 16S rRNA gene sequence similarity (>99.59%), but average nucleotide identity (ANI) and <I>in silico</I> DNA-DNA hybridization (DDH) values and multilocus sequence- and genome-based relatedness analyses suggested that they were divided into four different phylogenetic lineages. Relatedness analyses based on multilocus sequences, 1,194 core genes and whole-cell MALDI-TOF profiles supported that strains LMG 1590<SUP>T</SUP> and LMG 1591 (previously classified as the type strains of <I>A. pasteurianus</I> subsp. <I>ascendens</I> and <I>paradoxus</I>, respectively) and strain SLV-7<SUP>T</SUP> do not belong to <I>A. pasteurianus</I>. Strain SLV-7<SUP>T</SUP>, isolated from Korean traditional vinegar, shared low ANI (<91.0%) and <I>in silico</I> DDH (44.2%) values with all other <I>Acetobacter</I> type strains analyzed in this study, indicating that strain SLV-7<SUP>T</SUP> represents a new <I>Acetobacter</I> species. The phenotypic and chemotaxonomic analyses confirmed these results and therefore a new species named <I>Acetobacter oryzifermentans</I> sp. nov. is proposed with SLV-7<SUP>T</SUP> (=KACC 19301<SUP>T</SUP> =JCM 31096<SUP>T</SUP>) as the type strain. Strains LMG 1590<SUP>T</SUP> and LMG 1591 shared high ANI (99.4%) and <I>in silico</I> DDH (96.0%) values between them, but shared low ANI (<92.3%) and <I>in silico</I> DDH (<49.0%) values with other type strains analyzed in this study, indicating that strains LMG 1590<SUP>T</SUP> and LMG 1591 should be reclassified into a new single species that should be named <I>Acetobacter ascendens</I> sp. nov., comb. nov., with LMD 51.1<SUP>T</SUP> (=LMG 1590<SUP>T</SUP> =NCCB 51001<SUP>T</SUP>) as its type strain.</P>
Kim, Moon Young,Baik, Soon Koo,Yea, Chang Jin,Lee, Il Young,Kim, Hye Jung,Park, Kyong Won,Kim, Hearn Kook,Suk, Ki Tae,Kim, Jae Woo,Kim, Hyun Soo,Kwon, Sang Ok,Cha, Seung Hwan,Kim, Young Ju,Koh, Sang B Lippincott Williams Wilkins, Inc. 2009 European journal of gastroenterology & hepatology Vol.21 No.11
OBJECTIVE: Portal hypertension is closely associated with serious complications of cirrhosis, which contribute to bad prognosis. Hepatocellular carcinoma (HCC) and low serum sodium (SNa) are manifestations of end-stage liver disease and are associated with poor survival in decompensated cirrhosis patients. We aimed to determine the relationship between hepatic venous pressure gradient (HVPG) and the development of HCC or low SNa in decompensated alcoholic cirrhosis patients. METHODS: Child-Pugh scores, Model for End-Stage Liver Disease scores, and HVPG at baseline, and the development of HCC or low SNa (SNa <130 mEq/l) during follow-up were analyzed prospectively in 170 patients with decompensated alcoholic cirrhosis from December 1999 to January 2008 (mean follow-up period of 33.9±27.9 months). The predictive value of different risk factors for the development of HCC and low SNa and survival were investigated. RESULTS: Twenty-four patients developed HCC during the follow-up period. In the multivariate analysis, only baseline HVPG greater than 15 mmHg was an independent predictive factor for the development of HCC (relative risk=1.128, P<0.05) and which showed a significantly shorter time for the development of HCC on the Kaplan–Meier analysis. Twenty patients developed low SNa during follow-up. Initial HVPG was also an independent predictive factor for the new development of low SNa in the multivariate analysis (relative risk=1.169, P<0.05) and which also showed significantly shorter times for the development of low SNa on the Kaplan–Meier analysis. CONCLUSION: In decompensated alcoholic cirrhosis, HVPG may be a useful predictive factor for the development of HCC and low SNa.
Ok Hwan Cha,Cheol-Hoi Kim,Jun Seok Lee,Jong Pil Jeong,Joong Seo Park,Jandi Kim,정현,Eun-Kyung Suh 한국물리학회 2009 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.55 No.1
The temperature dependence of the electroluminescence spectra of InGaN/GaN multi-quantumwell light-emitting diodes (LEDs) has been investigated over a range of temperatures and currents. The presence of localization effects in the active InGaN layers can be deduced from the observed considerable blue shifts of the electroluminescence emission peak with increasing temperature in the low-temperature region. Exciton-phonon couplings in the active layer are also observed at low temperatures. The electroluminescence intensity was measured to determine the internal quantum efficiency of the LEDs as a function of temperature. When the temperature decreases from room temperature to 170~200 K, the electroluminescence intensity increases due to a reduction in the nonradiative recombination, and the internal quantum efficiency is improved. At T < 170 K, the electroluminescence intensity is reduced significantly. This reduction of the electroluminescence intensity mainly results from the low carrier capture efficiency in the active layer due to the high Mg activation energy, the electron-hole separation in multi quantum wells, and the high internal piezoelectric field. However, in the case of LEDs using a narrow barrier, the electroluminescence intensity is reduced slightly at low temperatures. Therefore, in order to measure the internal quantum efficiency by using the temperature dependence of the electroluminescence spectra in LEDs, one must carefully consider the effects of carrier injection at low temperature.
Kim, Su Jin,Bieganski, Tadeusz,Sohn, Young Bae,Kozlowski, Kazimierz,Sem?nov, Mikhail,Okamoto, Nobuhiko,Kim, Chi Hwa,Ko, Ah-Ra,Ahn, Geung Hwan,Choi, Yoon-La,Park, Sung Won,Ki, Chang-Seok,Kim, Ok-Hwa,Ni Springer-Verlag 2011 HUMAN GENETICS Vol.129 No.5
<P>Sclerosteosis and Van Buchem disease are related recessive sclerosing bone dysplasias caused by alterations in the SOST gene. We tested the hypothesis that craniodiaphyseal dysplasia (CDD) (MIM 122860), an extremely rare sclerosing bone dysplasia resulting facial distortion referred to as 'leontiasis ossea', could also be caused by SOST mutations. We discovered mutations c.61G>A (Val21Met) and c.61G>T (Val21Leu) two children with CDD. As these mutations are located in the secretion signal of the SOST gene, we tested their effect on secretion by transfecting the mutant constructs into 293E cells. Intriguingly, these mutations greatly reduced the secretion of SOST. We conclude that CDD, the most severe form of sclerotic bone disease, is part of a spectrum of disease caused by mutations in SOST. Unlike the other SOST-related conditions, sclerosteosis and Van Buchem disease that are inherited as recessive traits seem to be caused by a dominant negative mechanism.</P>