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Shuji Itakura,Kazuya Hattori,Kiwamu Umezawa 한국응용곤충학회 2018 Journal of Asia-Pacific Entomology Vol.21 No.1
A total of 114 microRNAs (miRNAs) were identified for Coptotermes formosanus workers, while a total of 97miRNAs were identified for Reticulitermes speratus workers, of which 91 were common in C. formosanus and R. speratus. While the relationship between miRNA expression levels in C. formosanus and R. speratus workers had astrong positive correlation, considerable difference in miRNA expressions between C. formosanus and R. speratuswas observed. Among the miRNAs up-regulated in C. formosanus workers, miR-11-3p showed the highest expressionincrease of 2.30 fold, while miR-13b-3p showed the highest expression increase of 5.16 fold among theup-regulated miRNAs in R. speratus workers. Workers of C. formosanus and R. speratus seem to use differentmiRNAs, miR-11-3p in C. formosanus or miR-13b-3p in R. speratus, to regulate homeodomain transcription factorgenes, araucan, caupolican, and nubbin, basic helix-loop-helix (bHLH) transcription factor genes, spineless and E(spl)m3-HLH, and zinc finger transcription factor gene, castor.
Aoyama, Yuki,Toriumi, Kazuya,Mouri, Akihiro,Hattori, Tomoya,Ueda, Eriko,Shimato, Akane,Sakakibara, Nami,Soh, Yuka,Mamiya, Takayoshi,Nagai, Taku,Kim, Hyoung-Chun,Hiramatsu, Masayuki,Nabeshima, Toshitak American College of Neuropsychopharmacology 2016 Neuropsychopharmacology Vol.41 No.2
<P>Cigarette smoking during pregnancy is associated with various disabilities in the offspring such as attention deficit/hyperactivity disorder, learning disabilities, and persistent anxiety. We have reported that nicotine exposure in female mice during pregnancy, in particular from embryonic day 14 (E14) to postnatal day 0 (P0), induces long-lasting behavioral deficits in offspring. However, the mechanism by which prenatal nicotine exposure (PNE) affects neurodevelopment, resulting in behavioral deficits, has remained unclear. Here, we report that PNE disrupted the proliferation of neuronal progenitors, leading to a decrease in the progenitor pool in the ventricular and subventricular zones. In addition, using a cumulative 5-bromo-2'-deoxyuridine labeling assay, we evaluated the rate of cell cycle progression causing the impairment of neuronal progenitor proliferation, and uncovered anomalous cell cycle kinetics in mice with PNE. Accordingly, the density of glutamatergic neurons in the medial prefrontal cortex (medial PFC) was reduced, implying glutamatergic dysregulation. Mice with PNE exhibited behavioral impairments in attentional function and behavioral flexibility in adulthood, and the deficits were ameliorated by microinjection of D-cycloserine into the PFC. Collectively, our findings suggest that PNE affects the proliferation and maturation of progenitor cells to glutamatergic neuron during neurodevelopment in the medial PFC, which may be associated with cognitive deficits in the offspring.</P>