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Oide, Shinichi,Liu, Jinyuan,Yun, Sung-Hwan,Wu, Dongliang,Michev, Alex,Choi, May Yee,Horwitz, Benjamin A.,Turgeon, B. Gillian American Society for Microbiology 2010 EUKARYOTIC CELL Vol.9 No.12
<B>ABSTRACT</B><P>Histidine kinase (HK) phosphorelay signaling is a major mechanism by which fungi sense their environment. The maize pathogen Cochliobolus heterostrophus has 21 HK genes, 4 candidate response regulator (RR) genes (<I>SSK1</I>, <I>SKN7</I>, <I>RIM15</I>, <I>REC1</I>), and 1 gene (<I>HPT1</I>) encoding a histidine phosphotransfer domain protein. Because most HKs are expected to signal through RRs, these were chosen for deletion. Except for pigment and slight growth alterations for <I>rim15</I> mutants, no measurable altered phenotypes were detected in <I>rim15</I> or <I>rec1</I> mutants. Ssk1p is required for virulence and affects fertility and proper timing of sexual development of heterothallic C. heterostrophus. Pseudothecia from crosses involving <I>ssk1</I> mutants ooze masses of single ascospores, and tetrads cannot be found. Wild-type pseudothecia do not ooze. Ssk1p represses asexual spore proliferation during the sexual phase, and lack of it dampens asexual spore proliferation during vegetative growth, compared to that of the wild type. <I>ssk1</I> mutants are heavily pigmented. Mutants lacking Skn7p do not display any of the above phenotypes; however, both <I>ssk1</I> and <I>skn7</I> mutants are hypersensitive to oxidative and osmotic stresses and <I>ssk1 skn7</I> mutants are more exaggerated in their spore-type balance phenotype and more sensitive to stress than single mutants. <I>ssk1</I> mutant phenotypes largely overlap <I>hog1</I> mutant phenotypes, and in both types of mutant, the Hog1 target gene, <I>MST1</I>, is not induced. <I>ssk1</I> and <I>hog1</I> mutants were examined in the homothallic cereal pathogen Gibberella zeae, and pathogenic and reproductive phases of development regulated by Ssk1 and Hog1 were found to mirror, but also vary from, those of C. heterostrophus.</P>
Yawen Guo,Jinyuan Chen,Shuyu Liu,Yali Zhu,Pengfei Gao,Kaizhou Xie 한국축산학회 2022 한국축산학회지 Vol.64 No.5
This study aimed to determine the effect of dietary supplementation with Acremonium terricolaculture (ATC) on the quality, conventional characteristics, and flavor substances of Hortobágygoose meat. A total of 720 one-day-old goslings were divided into four dietary treatments, eachconsisting of six cages of 30 goslings. The dietary conditions consisted of the control group andthree treatment groups supplemented with 3, 5, or 7 g/kg ATC. In male geese, supplementationwith 3 g/kg ATC elevated the crude ash (CA) content of the thigh muscle compared to the controlgroup, and the CA content of the pectoralis major was significantly elevated when geesewere supplemented with 5 g/kg ATC (p < 0.05). In females, compared with the control group,supplementation with 7 g/kg ATC enhanced the crude protein (CP) content of the pectoralismajor. Supplementation with 7 g/kg ATC also increased the crude fat (CF) content of the pectoralismajor in females as well as in both sexes; moreover, this supplementation dose increasedthe inosinic acid content of the thigh muscle in males and in both sexes. In contrast, supplementationwith 5 g/kg ATC decreased the pH of the thigh muscle at 12 h postmortem (p < 0.01). No significant changes in meat color, water loss rate, shear force, moisture content or aminoacid (AA) levels were observed after ATC supplementation (p > 0.05). Levels of saturated fattyacids (SFAs) and polyunsaturated FAs (PUFAs) in the pectoralis major and levels of SFAs,monounsaturated FAs (MUFAs), and PUFAs in the thigh muscle were not affected by the supplementation. Overall, ATC supplementation had positive effects on the pH, and CA, CP, CF,inosinic acid contents as well as on the FA composition of gosling meat. The optimal level ofATC supplementation was 7 g/kg in goslings from 1 to 70 days of age.
Research on Problems of the Loss of Distributed Power Grid Switch and Heat Dissipation
Rui Guo,Liaoyi Ning,Jinyuan Liu 보안공학연구지원센터 2016 International Journal of Grid and Distributed Comp Vol.9 No.9
At present, the technology of the distributed power grid gradually become mature. The focus of the study is from controlling power quality to reducing the loss of power grid, improving the network efficiency and reducing the cost. In the link of distributed generation grid connected power generation, the loss of the switching device is an important factor affecting the efficiency of grid connection. This paper puts forward a systematic solution to this problem. Firstly, as for topology structure of grid connected controller, aiming at the research on NPC, a traditional topology which is energy-saving and low loss, we present an improved NPC topology with a more continuous flow state. At the same time for this topology, the corresponding control strategies are designed in this paper. It makes the switching device of the grid connected controller have more freedom of switch and make the system can achieve the precise control of the switch tube ,which can achieve the balance of system device lose heat so as to solve the problem of local high temperature from inverter. The system is verified by grid connected test, the power adjustable PWM strategy can be used to achieve different working states without affecting the output current of the system. Through the comparison between light and heat sensors finds that the topology and control strategy proposed in this paper are successfully completed by the new hardware topology and control strategy to achieve the loss balance of the system switching devices.
Molybdenum Dioxide-Anchored Graphene Foam as a Negative Electrode Material for Supercapacitors
Xuemei Mu,Xiaozhi Liu,Ke Zhang,Jian Li,Jinyuan Zhou,ER-QING XIE,Zhenxing Zhang 대한금속·재료학회 2016 ELECTRONIC MATERIALS LETTERS Vol.12 No.2
Molybdenum dioxide nanoparticles of diameter 100 nm were anchoreduniformly to a three-dimensional (3D) graphene foam using an ultrasonicationassisteddeposition method. X-ray diffraction and Raman spectroscopyindicated that the molybdenum dioxide nanoparticles had a monoclinic crystalstructure. The 3D graphene/MoO2 nanoparticle composite showed excellentpseudocapacitive ability as its specific capacitance reached 404 F g−1 at a scanrate of 2 mV s−1 in the negative potential range, −1.0 to −0.2 V, in a neutralsolution. Overall, the 3D graphene/MoO2 nanoparticle composite has greatpotential as an anode material for the next generation of high-performancesupercapacitors.
Shi Si,Gu Huijie,Xu Jinyuan,Sun Wan,Liu Caiyin,Zhu Tong,Wang Juan,Gao Furong,Zhang Jieping,Ou Qingjian,Jin Caixia,Xu Jingying,Chen Hao,Li Jiao,Xu Guotong,Tian Haibin,Lu Lixia 생화학분자생물학회 2023 Experimental and molecular medicine Vol.55 No.-
Excessive osteoclast activation, which depends on dramatic changes in actin dynamics, causes osteoporosis (OP). The molecular mechanism of osteoclast activation in OP related to type 1 diabetes (T1D) remains unclear. Glia maturation factor beta (GMFB) is considered a growth and differentiation factor for both glia and neurons. Here, we demonstrated that Gmfb deficiency effectively ameliorated the phenotype of T1D-OP in rats by inhibiting osteoclast hyperactivity. In vitro assays showed that GMFB participated in osteoclast activation rather than proliferation. Gmfb deficiency did not affect osteoclast sealing zone (SZ) formation but effectively decreased the SZ area by decreasing actin depolymerization. When GMFB was overexpressed in Gmfb-deficient osteoclasts, the size of the SZ area was enlarged in a dose-dependent manner. Moreover, decreased actin depolymerization led to a decrease in nuclear G-actin, which activated MKL1/SRF-dependent gene transcription. We found that pro-osteoclastogenic factors (Mmp9 and Mmp14) were downregulated, while anti-osteoclastogenic factors (Cftr and Fhl2) were upregulated in Gmfb KO osteoclasts. A GMFB inhibitor, DS-30, targeting the binding site of GMFB and Arp2/3, was obtained. Biocore analysis revealed a high affinity between DS-30 and GMFB in a dose-dependent manner. As expected, DS-30 strongly suppressed osteoclast hyperactivity in vivo and in vitro. In conclusion, our work identified a new therapeutic strategy for T1D-OP treatment. The discovery of GMFB inhibitors will contribute to translational research on T1D-OP.