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장현철,김경식,방준현,최천진,엄정식,김응준,이루지 한국스포츠리서치 2004 한국 스포츠 리서치 Vol.15 No.2
The purpose of this study was to examine the perception and preference of skiers in a bid to provide ski resorts with useful information about successful positioning strategy. The subjects in this study were 9,722 skiers who purchased season tickets from N web site that sold ski season tickets. Their online purchasing route and page view were analyzed to find out what factors affected their purchase of ski season tickets. The findings of this study were as follows: First, 78 percent of the people investigated were make, and 22 percent were female. By age, the largest group was in their 20s(5,344 skiers), followed by those in their 30s(3,434), those in their 40s(631) and teenagers(229). Second, their page view rate with Yangji recorded highest with 24%, followed by Sungwoo Resort with 23% and Chonmasan with 13 percent. Third, by geographic area, the skiers from southern Gyeonggi province bought the largest number of tickets for the 2002/2003 season, followed by Gangweon province residents and northem Gyeonggi province dwellers. In the case of the 2003/2004 season, those from Gangweon provinece pruchased the most tickets, followed by southern Gyeonggi province dwellers and north Gyeonggi province residents.
( Ji Young Eum ),( Yeoung Kyun Kim ),( Eun Ji Park ),( Ju Hee Lee ),( Ji Eun Lee ),( Jin Ju Lim ),( Man Ho Choi ),( Hyun Hee Kim ) 물리치료재활과학회 2015 Physical therapy rehabilitation science Vol.4 No.1
Objective: Jump training helps increase the muscle power by improving the muscle strength and reaction time of the muscle in operation. The purpose of this study was to identify the effects of strengthening, stretching exercise and meditation on electromyographic (EMG) onset timing of rectus femoris and gastrocnemius muscle during vertical jump performance. Design: Cross-sectional study. Methods: Ten healthy adults (5 male and 5 female) who were familiar with the vertical jumping task and had no lower extremity injuries or any bone or joint disorders, were recruited for this study. Muscle onset timing was measured by surface EMG. After EMG onset timing were measured during performing three baseline vertical jump trials, strengthening and stretching exercises of the rectus femoris and gastrocnemius, and meditation were performed in random order. EMG onset timing was measured during vertical jump after intervention, respectively. EMG value was averaged for the three trials and analyzed using one-way repeated ANOVA. Results: During vertical jump, EMG onset timing of gastrocnemius was a significant difference after intervention (p<0.05), and then there was significantly faster in strengthening exercise than meditation (p<0.05). Conclusions: These results indicate the potential positive effect of performing strengthening exercise of the gastrocnemius before a jumping event. Future research is required to identify the effects of intervention over a long period.
Eum, Ji Young,Kim, Yeoung Kyun,Park, Eun Ji,Lee, Ju Hee,Lee, Ji Eun,Lim, Jin Ju,Choi, Man Ho,Kim, Hyun Hee korean Academy of Physical Therapy Rehabilitation 2015 Physical therapy rehabilitation science Vol.4 No.1
Objective: Jump training helps increase the muscle power by improving the muscle strength and reaction time of the muscle in operation. The purpose of this study was to identify the effects of strengthening, stretching exercise and meditation on electromyographic (EMG) onset timing of rectus femoris and gastrocnemius muscle during vertical jump performance. Design: Cross-sectional study. Methods: Ten healthy adults (5 male and 5 female) who were familiar with the vertical jumping task and had no lower extremity injuries or any bone or joint disorders, were recruited for this study. Muscle onset timing was measured by surface EMG. After EMG onset timing were measured during performing three baseline vertical jump trials, strengthening and stretching exercises of the rectus femoris and gastrocnemius, and meditation were performed in random order. EMG onset timing was measured during vertical jump after intervention, respectively. EMG value was averaged for the three trials and analyzed using one-way repeated ANOVA. Results: During vertical jump, EMG onset timing of gastrocnemius was a significant difference after intervention (p<0.05), and then there was significantly faster in strengthening exercise than meditation (p<0.05). Conclusions: These results indicate the potential positive effect of performing strengthening exercise of the gastrocnemius before a jumping event. Future research is required to identify the effects of intervention over a long period.
Hyun Ju Cha,Won Sik Eum,Gi Soo Youn,Jung Hwan Park,Hyeon Ji Yeo,Eun Ji Yeo,Hyun Jung Kwon,Lee Re Lee,Na Yeon Kim,Su Yeon Kwon,Yong-Jun Cho,Sung-Woo Cho,Oh-Shin Kwon,Eun Jeong Sohn,Dae Won Kim,Duk-Soo 생화학분자생물학회 2023 BMB Reports Vol.56 No.4
Thioredoxin-like protein 1 (TXNL1), one of the thioredoxin superfamilyknown as redox-regulator, plays an essential in maintainingcell survival via various antioxidant and anti-apoptoticmechanisms. It is well known that relationship between ischemiaand oxidative stress, however, the role of TXNL1 protein inischemic damage has not been fully investigated. In the presentstudy, we aimed to determine the protective role of TXNL1against on ischemic injury in vitro and in vivo using cellpermeable Tat-TXNL1 fusion protein. Transduced Tat-TXNL1inhibited ROS production and cell death in H2O2-exposedhippocampal neuronal (HT-22) cells and modulated MAPKsand Akt activation, and pro-apoptotic protein expression levelsin the cells. In an ischemia animal model, Tat-TXNL1 markedlydecreased hippocampal neuronal cell death and the activationof astrocytes and microglia. These findings indicate thatcell permeable Tat-TXNL1 protects against oxidative stress invitro and in vivo ischemic animal model. Therefore, we suggestTat-TXNL1 can be a potential therapeutic protein for ischemicinjury.
Kim, Hyun Jong,Choi, Ji Hyun,Hwang, Jung-Hwan,Kim, Kyong-Shim,Noh, Jung-Ran,Choi, Dong-Hee,Moon, Sung Je,Kim, Hyun-Yong,Kim, Sang-Woo,Choi, Sangho,Eum, Sang Mi,Bach, Tran The,Rho, Jaerang,Lee, Ju Yong UNKNOWN 2018 INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE Vol.42 No.5
<P><I>Melicope ptelefolia</I> has been traditionally used to treat rheumatism and fever. The present study aimed to investigate the therapeutic effect of 3,5-di-C-<I>β</I>-<SUB><SMALL>D</SMALL></SUB>-glucopyranosyl phloroacetophenone (βGP), a main component of <I>M. ptelefolia</I>, on rheumatoid arthritis (RA). A model of collagen-induced arthritis (CIA) was established in mice using the RAW 264.7 murine macrophage cell line and mouse embryonic fibroblasts (MEFs). The clinical scores of arthritis, swelling, histopathological findings, and micro-computed tomography in CIA mouse paws were assessed. The levels of anti-type II collagen antibody and cytokines were determined in the plasma and cell culture supernatant, respectively. Protein and gene expression levels were analyzed by western blot and reverse transcription-quantitative polymerase chain reaction analyses. βGP significantly decreased the gross arthritic scores of CIA mice and joint swelling, and decreased articular inflammation, cartilage degradation and bone erosion. However, βGP did not exert any effect on anti-type II collagen immunoglobulin G plasma levels or inflammatory cytokine expression in macrophages. βGP significantly suppressed the expression of interleukin-6 and leukemia inhibitory factor and decreased the phosphorylation of signal transducer and activator of transcription 3, and expression of receptor activator of nuclear factor-κB ligand in tumor necrosis factor-α-stimulated MEFs and in CIA mouse paws. Osteoclast-related gene expression was significantly reduced in CIA mouse paws. Taken together, βGP suppressed the development of RA by regulating the activation of synovial fibroblasts.</P>
이지현(Ji Hyun Lee),장민선(Min Sun Chang),양해조(Hae Jo Yang),엄향란(Hyang Lan Eum),최현진(Hyun Jin Choi),임수연(Soo Yeon Lim),홍윤표(Yoon Pyo Hong) 한국원예학회 2021 한국원예학회 학술발표요지 Vol.2021 No.10
최근 코로나 펜데믹으로 인한 신선 농산물 항공 수출길이 어려워지고, 2024년 수출 물류비 지원이 중단됨에 따라 선박이용 필요성이 대두되고 있다. 이에 본 연구에서는 딸기를 대상으로 CA 컨테이너 환경조건을 구명하고, 선박운송 중 발생하는 물러짐, 부패 등의 품질저하 문제를 보완하고자 하였다. 딸기는 2021년 3월에 수확한 것으로 홍성지역 ‘아리향’, 논산지역 ‘금실’, ‘킹스베리’, ‘비타베리’, ‘설향’, 담양지역 ‘죽향’ 등 총 6품종을 사용하였고, 기능성 1단 MA 용기를 일부 적용하였다. CA 컨테이너 온도는 4°C, 환경조건은 산소 5%, 이산화탄소 12% 수준으로 설정하였고, 모의수송 7일 후 상온유통 조건에서 품질변화를 관찰하였다. 대조구로는 일반 저장고를 4°C 설정하여 사용하였다. 모의수송 중 컨테이너 내부 CA 환경변화를 모니터링 한 결과, 산소 7.2%, 이산화탄소 10% 수준으로 유지되었으며 온도는 5.1~6.2°C 범위로 팔레트 위치별, 시간대별 온도편차가 있었다. 전반적으로 CA 컨테이너 처리 딸기 중량감소율이 낮았고, 특히 기능성 용기 딸기에서 현저히 낮았다. 경도는 CA 컨테이너 딸기가 높았고, 모의수송 중 다소 증가하는 경향을 보였으며 품종별로는 죽향 < 비타베리 < 킹스베리 < 금실 < 아리향 순으로 높았다. 당도, 산도, 호흡률은 CA 컨테이너 처리에 따른 유의적인 차이가 없었으나 부패, 물러짐은 CA 컨테이너 딸기에서 현저히 적어 상품성이 양호하였다. 전반적으로 CA 컨테이너 적용은 수출 딸기의 손실률 감소, 단단함과 향미 유지에 효과적이었다. 앞으로 수출 신선 농산물에 대한 품목별 CA 컨테이너 활용 상품화 기술 개발로 보다 안정된 선박수출이 가능하리라 판단된다.
SOHN, EUN JEONG,SHIN, MIN JEA,EUM, WON SIK,KIM, DAE WON,YONG, JI IN,RYU, EUN JI,PARK, JUNG HWAN,CHO, SU BIN,CHA, HYUN JU,KIM, SANG JIN,YEO, HYEON JI,YEO, EUN JI,CHOI, YEON JOO,IM, SEUNG KWON,KWEON, HA Spandidos Publications 2016 INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE Vol.38 No.1
<P>Oxidative stress-induced apoptosis is associated with neuronal cell death and ischemia. The NOL3 [nucleolar protein 3 (apoptosis repressor with CARD domain)] protein protects against oxidative stress-induced cell death. However, the protective mechanism responsible for this effect as well as the effects of NOL3 against oxidative stress in ischemia remain unclear. Thus, we examined the protective effects of NOL3 protein on hydrogen peroxide (H2O2)-induced oxidative stress and the mechanism responsible for these effects in hippocampal neuronal HT22 cells and in an animal model of forebrain ischemia using Tat-fused NOL3 protein (Tat-NOL3). Purified Tat-NOL3 protein transduced into the H2O2-exposed HT22 cells and inhibited the production of reactive oxygen species (ROS), DNA fragmentation and reduced mitochondrial membrane potential (Delta(Psi m)). In addition, Tat-NOL3 prevented neuronal cell death through the regulation of apoptotic signaling pathways including Bax, Bcl-2, caspase-2, -3 and -8, PARP and p53. In addition, Tat-NOL3 protein transduced into the animal brains and significantly protected against neuronal cell death in the CA1 region of the hippocampus by regulating the activation of microglia and astrocytes. Taken together, these findings demonstrate that Tat-NOL3 protein protects against oxidative stress-induced neuronal cell death by regulating oxidative stress and by acting as an anti-apoptotic protein. Thus, we suggest that Tat-NOL3 represents a potential therapeutic agent for protection against ischemic brain injury.</P>
Eum, Won Sik,Shin, Min Jea,Lee, Chi Hern,Yeo, Hyeon Ji,Yeo, Eun Ji,Choi, Yeon Joo,Kwon, Hyun Jung,Kim, Duk-Soo,Kwon, Oh Shin,Lee, Keun Wook,Han, Kyu Hyung,Park, Jinseu,Kim, Dae Won,Choi, Soo Young Elsevier 2019 Biochimie Vol.156 No.-
<P><B>Abstract</B></P> <P>Parkinson's disease (PD), a neurodegenerative disorder, is characterized by a loss of dopaminergic neurons in the substantia nigra (SN) of the brain and it is well known that the pathogenesis of PD is related to a number of risk factors including oxidative stress. Antioxidant 1 (ATOX1) protein plays a crucial role in various diseases as an antioxidant and chaperone. In this study, we determined whether Tat-ATOX1 could protect against 1-methyl-4-phenylpyridinium ion (MPP<SUP>+</SUP>)-induced SH-SY5Y cell death and in a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced animal model of PD. In the MPP<SUP>+</SUP> exposed SH-SY5Y cells, Tat-ATOX1 markedly inhibited cell death and toxicities. In addition, Tat-ATOX1 markedly suppressed the activation of Akt and mitogen activated protein kinases (MAPKs) as well as cleavage of caspase-3 and Bax expression levels. In a MPTP-induced animal model, Tat-ATOX1 transduced into brain and protected dopaminergic neuronal cell loss. Taken together, Tat-ATOX1 inhibits dopaminergic neuronal death through the suppression of MAPKs and apoptotic signal pathways. Thus, Tat-ATOX1 represents a potential therapeutic protein drug candidate for PD.</P> <P><B>Highlights</B></P> <P> <UL> <LI> Tat-ATOX1 transduces into SH-SY5Y cells and inhibited MPP<SUP>+</SUP>-induced cell death. </LI> <LI> Tat-ATOX1 suppressed the activation of Akt and MAPKs in MPP<SUP>+</SUP> exposed SH-SY5Y cells. </LI> <LI> Tat-ATOX1 inhibited dopaminergic neuronal cell loss in MPTP-induced animal model. </LI> <LI> Tat-ATOX1 represent a potential therapeutic agent for PD. </LI> </UL> </P>