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OPENING AN EMBASSY IN SEOUL: SOME REFLECTIONS
GORDON LONGMUIR 계명대학교 한국학연구원 2005 Acta Koreana Vol.8 No.1
Though Canada established formal diplomatic relations with the Republic of Korea in 1964, there was no embassy in Seoul until 1973. That year D. Gordon Longmuir was dispatched to Seoul to help open that embassy and serve as First Secretary and Consul, as well as Chargé d’Affaires until a resident ambassador arrived early in 1974. He stayed in Seoul until 1976. Among the more pressing duties of the embassy while he was stationed there was ensuring adequate safeguards for a nuclear reactor Korea was considering buying from Atomic Energy of Canada Limited (AECL). At first Canada, somewhat distrustful of President Park Chung Hee, was uncertain whether or not to provide Park with such a nuclear power plant. However, after the ROK agreed to ratify the Nuclear Non-proliferation Treaty, the deal was signed and Korea eventually went on to purchase a total of four Canadian-designed CANDU nuclear reactors. As a representative of the Canadian government, Longmuir was in the audience on August 15, 1974, when a disgruntled Korean-Japanese attempted to assassinate Park but shot and killed Park’s wife instead. He also watched as, under Park’s leadership, the Korean economy began its rapid march to modernization while the government resisted pressure for democratization. The embassy occasionally had to intervene on behalf of some activist Canadian missionaries who felt that urban workers were being asked to pay too heavy a price for Korea’s economic progress.
The effect of glucocorticoids on bone and muscle
Gordon L. Klein 대한골다공증학회 2015 Osteoporosis and Sarcopenia Vol.1 No.1
This article examines the current knowledge of the effects of both exogenous and endogenous glucocorticoids on bone and muscle. It demonstrates the similarity of effects of supraphysiologic loads of glucocorticoids regardless of whether they enter the body in the form of medication or are manufactured by the body in response to stimuli such as inflammation. The effects of endogenous glucocorticoids and the systemic inflammatory response resulting from pediatric burn injury are compared and the difficulty in sorting out which of the two factors is responsible for the ultimate effects on bone and muscle is pointed out. The focus then switches to the body's response to the influence of both glucocorticoids and inflammatory cytokines and evidence supporting a common pathway of response to oxidative damage caused by both is discussed. Current recommended medical management of glucocorticoid-induced bone and muscle loss is discussed and the failure to reconcile current management with known mechanisms is highlighted.