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Study of Elastic Moduli of Sintered Low Alloy Steels by Acoustic Pulse Method
Hirose Norimitsu,Oouchi Kazuya,Fujiki Akira,Asami Junichi 한국분말야금학회 2006 한국분말야금학회 학술대회논문집 Vol.2006 No.1
The influence of porosity (P) on Young's modulus (E) and Poisson's ratio of sintered steels produced from four types of steel powders was investigated. The values of E and depend mainly on the value of P, and those were a little affected by alloying elements. The relationships between E, , and P were described as following equations: and , where subscript 0 means P = 0, and and are empirical constants. These approximate equations showed good agreement with empirical results.
Yoshinori Hayakawa,Zhongrui Duan,Miki Yadake,Yasuyo Yamaoka,Rie Inatsugi,Yuki Fujiki,Akira Oikawa,Kazuki Saito,Ikuo Nishida 한국식물학회 2015 Journal of Plant Biology Vol.58 No.3
The apetala3 (ap3)-like homeotic mutation (ap3-HM) is recognized among pD991-AP3-derived Arabidopsis thaliana T-DNA-tagged lines carrying the -448 to +47 region of AP3 in their T-DNA. In the corresponding mutant lines for CTP:phosphorylcholine cytidylyltransferase genes, cct1-1 and cct2 (Inatsugi et al. 2009), some flowers of cct1-1 (F4) and many flowers of cct1-1 cct2 (F3) showed ap3-HM, and all flowers of cct1-1 (F5) and cct1-1 cct2 (F4) became increasingly homeotic. In contrast, cct2 flowers were normal for all generations tested. These results demonstrated that ap3-HM is linked to the cct1-1 allele and is enhanced by the cct2 allele. The ap3-HM in cct mutants was inversely correlated with AP3 transcript levels in enriched flower buds. Bisulfite sequencing revealed severe methylation within endogenous AP3 promoter regions in cct1-1 (F3; -317 to -2) and cct1-1 cct2 (F3; -473 to -2), but wild-type (Wassilevskaja) and cct2 plants showed no corresponding methylation. The ap3-HM in cct1-1 cct2 mutants was fully rescued by expressing a PISTILLATA promoter–AP3 construct, and was better alleviated in the F1 offspring of a cross with the CCT1-overexpressing mutant cct1-2 (Columbia) than with the wild type. We discuss possible links between expression of CCT and suppression of ap3-HM.