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Lee, Seonmin,Lee, Seon-Jin,Coronata, Anna A.,Fredenburgh, Laura E.,Chung, Su Wol,Perrella, Mark A.,Nakahira, Kiichi,Ryter, Stefan W.,Choi, Augustine M.K. Mary Ann Liebert 2014 Antioxidants & redox signaling Vol.20 No.3
<P>Sepsis, a systemic inflammatory response to infection, represents the leading cause of death in critically ill patients. However, the pathogenesis of sepsis remains incompletely understood. Carbon monoxide (CO), when administered at low physiologic doses, can modulate cell proliferation, apoptosis, and inflammation in pre-clinical tissue injury models, though its mechanism of action in sepsis remains unclear.</P>