http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
S. Entezari,I. Chitsaz,S. Kazemzadeh Hanani,M. Monemi 대한기계학회 2022 JOURNAL OF MECHANICAL SCIENCE AND TECHNOLOGY Vol.36 No.2
Most of the vehicle pollutants during emission tests are raised from catalyst inefficiency during cold start. Catalysts usually convert harmful emissions only when their temperature reaches around 250 °C to 350 °C. In this research, the vortex tube is implemented to recover the waste heat energy of exhaust gas during the cold start to improve catalyst heating. The experiments are conducted on the turbocharged direct-injection gasoline engine to extract the boundary conditions of numerical simulations. Numerical simulations are performed to evaluate the effects of different hot exhaust mass fractions on the flow regime and waste heat recovery. The results reveal that the level of turbulence inside the vortex tube increases for higher hot mass fractions that lead to lower exhaust temperature on the hot side. By implementing the vortex tube, the maximum temperature at the hot exhaust is related to 20 % of the hot mass fraction and after that, the hot exhaust temperature decreases. By implementing the vortex tube, gas temperature before the catalyst is reached to 658 K at 52.3 % hot mass fraction which shows 48 K increase in exhaust temperature before the catalyst. At this point, 300 W heat is transferred to the exhaust gas that improves transient cold start time.
Reza Entezari Heravi,Farzin Hadizadeh,Mojtaba Sankian,Jalil Tavakol Afshari,Javad Behravan 한국유전학회 2012 Genes & Genomics Vol.34 No.2
Cyclooxygenase-2 (COX-2) inhibitors including celecoxib inhibit cell growth and induce apoptosis in cancer cells. In this study, the relation of Bax (an apoptosis promoter) to Bcl-2(an apoptosis inhibitor) ratio with the apoptosis co-ordination enzyme, caspase-3 was investigated in correlation with the treatment of 4,5-bisaryl imidazolyl imidazoles as novel selective COX-2 inhibitors in Caco-2 colorectal cancer cells . Recently, the organic reactions under microwave irradiation attracted attention of scientists due to their high reaction rate,mild reaction conditions and the formation of clean products. Therefore, a microwave-assisted method was used to synthesize our compounds. The effects of these COX-2 inhibitors on the proliferation of Caco-2 cells were evaluated by MTT assay. cDNA microarray and clustering analysis were used to evaluate effects of our synthetic compounds on gene expression pattern of 112 genes involved in apoptosis pathways. Bax, Bcl-2 and caspase-3 mRNA expression and their relationship were analyzed by quantitative real-time PCR. Results indicated that proliferation of Caco-2 cells after treatment with 4,5-bisaryl imidazolyl imidazoles on Caco-2 cells were time and dose dependent. We conclude that increase in Bax/Bcl-2ratio leads to an up-regulation in caspase-3 mRNA expression.
( Morteza Entezari ),( Alireza Ramezani ),( Mehdi Yaseri ) 대한안과학회 2012 Korean Journal of Ophthalmology Vol.26 No.2
In a clinical case series, 5 patients with not-resolved central serous choroidoretinopathy (CSC) lasting more than 1 year received one intravitreal bevacizumab injection (IVB, 1.25 mg) injection. All patients underwent a through ophthalmic examination 1 day, 1 week, and 1, 2, and 6 months after the injection. Best corrected visual acuity (BCVA) and central macular thickness were compared before and after treatment by optical coherence tomography. Mean BCVA was improved significantly (p=0.020) from 0.60±0.25 to 0.50±0.18 and 0.29±0.19 logarithm of minimum angle of resolution at 6 and 18 weeks, respectively. Central macular thickness was also decreased significantly (p=0.010) from 370±65 to 208±23 μm at 4 months. No recurrence was occurred during follow-up. IVB injection may have beneficial effect in the treatment of refractory CSC.
Amirijavid, Shaghayegh,Entezari, Maliheh,Movafagh, Abolfazl,Hashemi, Mehrdad,Mosavi-Jarahi, Alireza,Dehghani, Hossein Asian Pacific Journal of Cancer Prevention 2016 Asian Pacific journal of cancer prevention Vol.17 No.no.sup3
TRAIL, tumor necrosis factor (TNF)-related apoptosis-inducing ligand belongs to one of important cytokine superfamilIES, tumor necrosis factor ($TNF{\alpha}$). TRAIL-2 receptor agonists activate several cell signaling pathways in cells in different manners and could lead to apoptosis or necrosis. Agonistic egg yolk antibodies like IgY which have been developed in a selective manner could activate TRAIL death receptors such as TRAIL-2 (DR5) and thus apoptosis signaling. We here investigated induction of apoptosis in human breast cancer cells (MCF7 cell line) by an IgY produced against an 21 aminoacid epitope of the human TRAIL-2 receptor. As the first step a small peptide of 21 aminoacids choosen from the extracellular domain of DR5 protein was produced with a peptide synthesizer. After control assays and confirmation of the correct amino acid sequence, it was injected to hens immunized to achieve high affinity IgYs. At the next step, the produced IgYs were extracted and examined for specificity against DR5 protein by ELISA assay. Subsequently, the anticancer effect of such IgYs was determined by MTT assay in the MCF7 human breast cancer cell line. The produced peptides successfully immunized hens and the produced antibodies which accumulated in egg yolk specifically recognized the DR5 protein. IgYs exerted significant toxicity and killed MCF7 cells as shown by MTT assay.
Power-aware performance analysis of self-adaptive resource management in IaaS clouds
Ataie, Ehsan,Entezari-Maleki, Reza,Etesami, Sayed Ehsan,Egger, Bernhard,Ardagna, Danilo,Movaghar, Ali North-Holland 2018 Future generations computer systems Vol.86 No.-
<P><B>Abstract</B></P> <P>In this paper, Stochastic Activity Networks (SANs) are used to model and evaluate the performance and power consumption of an Infrastructure-as-a-Service (IaaS) cloud. The proposed SAN model is scalable and flexible, yet encompasses some details of an IaaS cloud, such as Virtual Machine (VM) provisioning, VM multiplexing, and failure/repair behavior of VMs. Using the proposed SAN, a power-aware self-adaptive resource management scheme is presented for IaaS clouds that automatically adjusts the number of powered-on Physical Machines (PMs) regarding variable workloads in different time intervals. The proposed scheme respects user-oriented metrics by avoiding Service Level Agreement (SLA) violations while taking provider-oriented metrics into consideration. The behavior of the proposed scheme is analyzed when the arriving workload changes, and then its performance is compared with two non-adaptive baselines based on diverse performance and power consumption measures defined on the system. A validation of the proposed SAN model and the resource management scheme against an adapted version of the CloudSim framework is also presented.</P> <P><B>Highlights</B></P> <P> <UL> <LI> An analytical model is proposed for Infrastructure-as-a-Service (IaaS) clouds taking several details of such systems into consideration. </LI> <LI> A self-adaptive power-aware and Service Level Agreement (SLA)-aware resource management scheme is presented for cloud systems. </LI> <LI> The presented scheme adjusts the number of powered-on Physical Machines (PMs) according to the input workload. </LI> <LI> A validation of the proposed model and scheme against the CloudSim framework is presented. </LI> </UL> </P>
Amirijavid, Shaghayegh,Entezari, Maliheh,Movafagh, Abolfazl,Hashemi, Mehrdad,Mosavi-Jarahi, Alireza,Dehghani, Hossein Asian Pacific Journal of Cancer Prevention 2016 Asian Pacific journal of cancer prevention Vol.17 No.no.sup3
Cancer causes cells to avoid death while being the second cause of death in the world itself. Damaged cells in the absence of apoptosis will increasingly amplify their inefficient genome. Of the two main apoptosis inducing pathways in cells, the first has p53 protein as the main initiating factor in the cascade. According to research results this protein s mutated in 50% of cancers and sointerest has cooncentrated on the second pathway that features death receptors. Among these receptors TRAIL1/DR5 is especially expressed in cancer cells. So targeting such receptors can initiate the apoptotic cascade in cells. Interestingly by substitution of activating ligands with antibodies as agonists, we could efficiently turn on the apoptosis pathway. First of all, three small peptides from the DR5 protein extracellular domain were synthesized and injected with two different kind of adjuvants (Fround and liposomal encapsulation) separately into mice at 15 day intervals. As a result, liposomal peptides induced the immune system more efficient than Frounds adjuvant and at the end point the antibodies which were obtained from liposomal peptide injection induced much more effective death. Liposomal formol could be used as an adjuvant in immunization utilizing small peptides. They carry, protect and deliver peptides very efficiently. In addition, small peptides of a certain size from the extracellular domain of DR5 proteins not only can induce immune system but also produce antibodies playing a remarkable anti-cancer roles against breast cancer cells (MCF-7).