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줄넘기운동 참여정도와 운동 만족도 및 신체적 자기개념의 관계
김충곤(Chung Gon Kim),이병규(Byung Gyu Lee),김현우(Hyun Woo Kim),양웅비(Woong Bi Yang) 한국체육교육학회 2013 한국체육교육학회지 Vol.18 No.2
본 연구의 목적은 초등학생을 대상으로 줄넘기운동 참여 정도와 줄넘기 운동만족도 및 신체적 자기개념 간의 인과 관계를 규명함으로써, 줄넘기운동이 초등학교 체육수업이나 방과후 프로그램 적용 가능성 및 줄넘기운동 권장에 대한 타당한 근거를 제시하는데 있다. 본 연구는 유의표 집(purposive sampling)을 이용하여 광주광역시 초등학교 4개교의 표본을 추출하여 총 733명을 연구 대상자로 선정하였다. 수집된 자료는 SPSS16와 Amos16을 이용하여 상관분석, 중다회귀분석, 공변량 구조분석 등의 통계분석을 활용하였다. 이와 같은 과정을 통하여 다음과 같은 결론을 도출하였다. 첫째, 줄넘기운동의 참여 정도는 운 동 만족도에 영향을 미친다. 둘째, 운동 만족도는 신체적 자기개념에 영향을 미친다. 셋째, 줄넘기운동의 참여 정도는 ‘자기 존중 감’을 제외한 모든 요인에서 신체적 자기개념에 영향을 미친다. 넷째, 줄넘기 참여 정도와 운동 만족도, 신체적 자기개념 간에는 인과적 관계가 있다. The purpose of this study is to provide the reasonable evidence of the applicability of rope-skipping exercise in elementary school physical education class or after-school class, and rope-skipping exercise recommendation by analyzing the casual relationship among participation degree of rope-skipping exercise on elementary school students, exercise satisfaction and physical self-concept. The purposive sampling was used for this study. Four elementary schools in Gwangju are sampled and 733 students are chosen as participants. SPSS16 and Amos16 was used to analyze collected materials and statistical analysis such as correlation analysis, multiple regression analysis and analysis of covariance structure were used. The conclusions based on the study were as follows: First, participation degree of skipping exercise had positive influence on exercise satisfaction. Second, exercise satisfaction had influence on physical self-concept. Third, participation degree of rope-skipping exercise had positive influence on physical self-concept in every factors except self-respect. Fourth, there is a casual relationship among participation degree of rope-skipping exercise, exercise satisfaction and physical self-concept.
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정비웅,김준성,송홍엽,Chung Bi-Woong,Kim Joon-Sung,Song Hong-Yeop 한국통신학회 2006 韓國通信學會論文誌 Vol.31 No.1c
LDPC 부호의 높은 부호화 복잡도는 구조적인 패리티 검사 행렬의 설계로 해결할 수 있다. 패리티 검사 행렬을 같은 유형의 블록으로 구성한다면 복호화기의 구현이 간단해지고 구조적 복호화가 가능하며 LDPC 부호를 저장하는데 필요한 메모리를 줄일 수 있는 장점이 있다. 본 논문에서는 부행렬 단위의 girth 조건과 PEG 알고리즘, 비트 노드의 connectivity를 이용하여 부행렬이 순환행렬이나 영행렬로 구성되는 짧은 길이를 갖는 구조적 LDPC 부호의 생성 알고리즘을 제안하였다. 이 알고리즘으로 생성된 부호는 구조적 제한이 없이 생성된 부호에 비하여 낮은 SNR에서는 비슷한 성능을, 높은 SNR에서는 더 좋은 성능을 내는 것을 모의 실험을 통해 확인하였다. The high encoding complexity of LDPC codes can be solved by designing structured parity-check matrix. If the parity-check matrix of LDPC codes is composed of same type of blocks, decoder implementation can be simple, this structure allow structured decoding and required memory for storing the parity-check matrix can be reduced largely. In this parer, we propose a construction algorithm for short block length structured LDPC codes based on girth condition, PEG algorithm and variable node connectivity. The code designed by this algorithm shows similar performance to other codes without structured constraint in low SNR and better performance in high SNR than those by simulation
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Da Yeon Kim,Seok Yun Jung,Yeon Ju Kim,Songhwa Kang,Ji Hye Park,Seung Taek Ji,Woong Bi Jang,Shreekrishna Lamichane,Babita Dahal Lamichane,Young Chan Chae,Dongjun Lee,Joo Seop Chung,Sang-Mo Kwon 대한생리학회-대한약리학회 2018 The Korean Journal of Physiology & Pharmacology Vol.22 No.2
Tumor undergo uncontrolled, excessive proliferation leads to hypoxic microenvironment. To fulfill their demand for nutrient, and oxygen, tumor angiogenesis is required. Endothelial progenitor cells (EPCs) have been known to the main source of angiogenesis because of their potential to differentiation into endothelial cells. Therefore, understanding the mechanism of EPC-mediated angiogenesis in hypoxia is critical for development of cancer therapy. Recently, mitochondrial dynamics has emerged as a critical mechanism for cellular function and differentiation under hypoxic conditions. However, the role of mitochondrial dynamics in hypoxia-induced angiogenesis remains to be elucidated. In this study, we demonstrated that hypoxiainduced mitochondrial fission accelerates EPCs bioactivities. We first investigated the effect of hypoxia on EPC-mediated angiogenesis. Cell migration, invasion, and tube formation was significantly increased under hypoxic conditions; expression of EPC surface markers was unchanged. And mitochondrial fission was induced by hypoxia time-dependent manner. We found that hypoxia-induced mitochondrial fission was triggered by dynamin-related protein Drp1, specifically, phosphorylated DRP1 at Ser637, a suppression marker for mitochondrial fission, was impaired in hypoxia timedependent manner. To confirm the role of DRP1 in EPC-mediated angiogenesis, we analyzed cell bioactivities using Mdivi-1, a selective DRP1 inhibitor, and DRP1 siRNA. DRP1 silencing or Mdivi-1 treatment dramatically reduced cell migration, invasion, and tube formation in EPCs, but the expression of EPC surface markers was unchanged. In conclusion, we uncovered a novel role of mitochondrial fission in hypoxia-induced angiogenesis. Therefore, we suggest that specific modulation of DRP1-mediated mitochondrial dynamics may be a potential therapeutic strategy in EPC-mediated tumor angiogenesis.
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Kim, Da Yeon,Jung, Seok Yun,Kim, Yeon Ju,Kang, Songhwa,Park, Ji Hye,Ji, Seung Taek,Jang, Woong Bi,Lamichane, Shreekrishna,Lamichane, Babita Dahal,Chae, Young Chan,Lee, Dongjun,Chung, Joo Seop,Kwon, Sa The Korean Society of Pharmacology 2018 The Korean Journal of Physiology & Pharmacology Vol.22 No.2
Tumor undergo uncontrolled, excessive proliferation leads to hypoxic microenvironment. To fulfill their demand for nutrient, and oxygen, tumor angiogenesis is required. Endothelial progenitor cells (EPCs) have been known to the main source of angiogenesis because of their potential to differentiation into endothelial cells. Therefore, understanding the mechanism of EPC-mediated angiogenesis in hypoxia is critical for development of cancer therapy. Recently, mitochondrial dynamics has emerged as a critical mechanism for cellular function and differentiation under hypoxic conditions. However, the role of mitochondrial dynamics in hypoxia-induced angiogenesis remains to be elucidated. In this study, we demonstrated that hypoxia-induced mitochondrial fission accelerates EPCs bioactivities. We first investigated the effect of hypoxia on EPC-mediated angiogenesis. Cell migration, invasion, and tube formation was significantly increased under hypoxic conditions; expression of EPC surface markers was unchanged. And mitochondrial fission was induced by hypoxia time-dependent manner. We found that hypoxia-induced mitochondrial fission was triggered by dynamin-related protein Drp1, specifically, phosphorylated DRP1 at Ser637, a suppression marker for mitochondrial fission, was impaired in hypoxia time-dependent manner. To confirm the role of DRP1 in EPC-mediated angiogenesis, we analyzed cell bioactivities using Mdivi-1, a selective DRP1 inhibitor, and DRP1 siRNA. DRP1 silencing or Mdivi-1 treatment dramatically reduced cell migration, invasion, and tube formation in EPCs, but the expression of EPC surface markers was unchanged. In conclusion, we uncovered a novel role of mitochondrial fission in hypoxia-induced angiogenesis. Therefore, we suggest that specific modulation of DRP1-mediated mitochondrial dynamics may be a potential therapeutic strategy in EPC-mediated tumor angiogenesis.
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