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RISS 인기검색어
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- 저자 : BRAUN J. E. (검색결과 3 건)
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BRAUN J. E.,KEEIN S. A.,MITCHELL J. W. 대한설비공학회 1983 설비저널 Vol.12 No.2
논문은 물을 저장물질로 사용하는 공간난방에 있어서의 seasonal storage의 몇 가지 중요한 문제연구하고 있다. 집열기 면적과 저장체적 그리고 시스템 성능간의 관계를 transient simulation program(TRNSYS)을 사용하여 조사하였다. 여기서 seasonal storage의 가장 일어나기 쉬운 상태가 나타내어지는데 seasonal storage system의 설계에 특히 역점을 두고 있다. 이러한 설계방법은 몇 일간에서 seasonal storage에 이르는 seasonal pacity (저장용량)에 대하여 적용되어진다. 비용과 관련하여 이러한 설계방법은 seasonal storage system 경제성 (economic viability)을 추정하는데 유용할 것이다. 또한 시스템 설계에서 부하 열기의 크기 탱크단열 집열기 경사 매년 기후변화의 중요성이 조사되고 있다.
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Potato lipoxygenase genes associated with development and pathogen defense response
Kolomiets, Michael V.,Chen, Hao,Braun, E.J.,Gladon, Richard J.,Hannapel, David J. 영남대학교 마늘연구소 2001 의성군 ·영남대학교 관 ·학협동, 협약 조인식 및 심포지움 Vol.2001 No.-
Plant lipoxygenases (LOXs) are a functionally diverse class of dioxygenases implicated in physiological processes such as growth, senescence, and stress-related responses. LOXs incorporate oxygen into their fatty acid substrates and produce hydroperoxide fatty acids that are precursors of jasmonic acid and related compounds. Here we report the involvement of a potato class-1 LOX, POTLX-1, in the control of tuber growth and a unique LOX, POTLX-3, in defense responses against pathogens. RNA hybridization analysis showed that accumulation of Lox1-class transcripts was specific to developing tubers and that mRNA accumulation correlated positively with tuber initiation and growth. Suppression mutants produced by expressing antisense coding sequence of the tuber LOX, POTLX-1, exhibited a significant reduction in LOX activity in stolons and tubers. The suppression of LOX activity correlated with reduced tuber yield, decreased average tuber size, and a disruption of tuber formation. POTLX-3 mRNA accumulation was induced specifically in potato leaves infected with either virulent or avirulent strains of Phytophthora infestans, the causal agent of late blight. During the resistance response, POTLX-3 was induced within 6 h, increased steadily through 24 h, and its mRNA continued to accumulate for a week after inoculation. In contrast, when a plant was susceptible to P. infestans, induction of mRNA accumulation in response to inoculation was inconsistent and delayed. LOX activity assayed during an incompatible interaction in leaves peaked three days earlier than during a compatible interaction. POTLX-3 mRNA accumulation also was induced during hypersensitive response development caused by the incompatible pathogen Pseudomonas syringae pv. phaseolicola. Our results show that LOXs of potato function in diverse roles in both development and in defense responses against pathogen infection.
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Mutations of CEP83 Cause Infantile Nephronophthisis and Intellectual Disability
Failler, M.,Gee, H.,Krug, P.,Joo, K.,Halbritter, J.,Belkacem, L.,Filhol, E.,Porath, Jonathan D.,Braun, Daniela A.,Schueler, M.,Frigo, A.,Alibeu, O.,Masson, C.,Brochard, K.,Hurault de Ligny, B.,Novo, R University of Chicago Press [etc.] 2014 American journal of human genetics Vol.94 No.6
Ciliopathies are a group of hereditary disorders associated with defects in cilia structure and function. The distal appendages (DAPs) of centrioles are involved in the docking and anchoring of the mother centriole to the cellular membrane during ciliogenesis. The molecular composition of DAPs was recently elucidated and mutations in two genes encoding DAPs components (CEP164/NPHP15, SCLT1) have been associated with human ciliopathies, namely nephronophthisis and orofaciodigital syndrome. To identify additional DAP components defective in ciliopathies, we independently performed targeted exon sequencing of 1,221 genes associated with cilia and 5 known DAP protein-encoding genes in 1,255 individuals with a nephronophthisis-related ciliopathy. We thereby detected biallelic mutations in a key component of DAP-encoding gene, CEP83, in seven families. All affected individuals had early-onset nephronophthisis and four out of eight displayed learning disability and/or hydrocephalus. Fibroblasts and tubular renal cells from affected individuals showed an altered DAP composition and ciliary defects. In summary, we have identified mutations in CEP83, another DAP-component-encoding gene, as a cause of infantile nephronophthisis associated with central nervous system abnormalities in half of the individuals.
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