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김지현(Jihyeon Kim),김진호(Jin-Ho Kim),김효식(Hyo-Sik Kim),류재홍(Jae-Hong Ryu),강석환(Suk-Hwan Kang),이예지(Ye-Jee Lee),이수출(Soo-Chool Lee) 한국에너지기후변화학회 2020 에너지기후변화학회지 Vol.15 No.2
Combustion characteristics of fuel oils such as bio heavy oil, pyrolysis oil, and ion refined oil were investigated to reserve basic data in terms of designing a reactor using fuel oil and selecting operating conditions. For the analysis of combustion characteristics, fuel oil was combusted with a thermogravimetric analyzer (TGA) under non-isothermal heating conditions (5 °C/min, 10 °C/min, 20 °C/min) in the air within the range of 25 °C to 945 °C. The activation energy and reaction order for fuel oil combustion have been kinetically interpreted using various methods such as the Kissinger, Chatterjee-Conrad, Coats-Redfern or Freeman-Carroll method. In this study, the activation energy of the three fuel oils was less than 50 kJ/mol, and Kissingers method with a reaction order of 1 showed an activation energy of less than 7.5. As a result, the activation energy and reaction order for the combustion characteristics of the three fuel oils were obtained for Freeman-Carrolls method among several interpretation methods. Through this, the Freeman-Carroll method was commonly applied according to the type of fuel oil (bio heavy oil, pyrolysis oil, ion refined oil), and the Kissinger method was limited to pyrolysis oil within a certain range. Through this study, it was possible to classify an appropriate analysis method according to the type of fuel oil.
발효산삼배양근농축액의 산화방지 효과 및 과산화수소로 유발된 PC12 세포독성 보호효과
최여옥(Yeo Ok Choi),김유리(Yu-Ri Kim),신승용(Seung-Yong Shin),이재근(Jae Geun Lee),김철중(Chul Joong Kim),이예지(Ye ji Lee),강병주(Byeongju Kang),김관수(Gwansu Kim),최지은(Jee Eun Choi),한범석(Beom-Seok Han) 한국식품과학회 2018 한국식품과학회지 Vol.50 No.4
대부분의 신경퇴행성질환은 산화스트레스의 영향을 받는다. 따라서 본 연구에서는 진세노사이드 Rg5와 Rk1을 함유하고 있는 발효산삼배양근 농축액인 HLJG0701의 산화방지 효능을 확인하였다. 그 결과, HLJG0701는 효과적인 DPPH와 ABTS 라디칼제거능을 나타냈으며(IC<SUB>50</SUB>: 16배, 4배 희석액), 황산철(II) 유도군의 지방질 과산화물 발생 비교 시, 용량의존적으로 억제됨을 확인하였다(8배 희석액: 2.3 nM, 4배 희석액: 1.5 nM). HLJG0701의 세포 보호 효과를 MTT, LDH 분석으로 평가한 결과, 8, 16, 32, 64배 희석액의 HLJG0701를 처리한 경우 70, 53, 35, 26%의 세포생존율이 증가됨을 확인하였다. LDH 방출 측정 결과, H₂O₂로 인해 3.6배까지 증가한 LDH 활성은 8배 희석액의 HLJG0701 처리시 1.3배 수준까지 감소하였다. 또한 HLJG0701가 PC12세포에 발생한 산화스트레스 DCF-DA 분석(16배 희석액: 50% ROS 억제, 4배 희석액: 68% ROS 억제), TBARS (16배 희석액: 50.7% 감소, 4배 희석액: 46.5% 감소), GPx (16배 희석액: 133.3% 증가, 4배 희석액: 227.3% 증가), 그리고 SOD (16배 희석액: 118.2% 증가, 4배 희석액: 218.2% 증가) 분석으로 확인하였다. 위 결과는 HLJG0701가 산화방지 효능을 매개로 신경세포를 보호함으로써 신경퇴행성질환을 예방할 수 있는 가능성 있는 후보물질이 될 수 있다는 것을 의미하며, 임상시험 등 다양한 연구가 충분하지 않은 상태이므로 앞으로 지속적인 연구가 필요할 것으로 사료된다. Most neurodegenerative diseases are known to be influenced by oxidative stress. We investigated the antioxidative activity of the concentrate of fermented wild ginseng root culture (HLJG0701) containing ginsenosides Rg5 and Rk1. HLJG0701 showed effective DPPH and ABTS radical scavenging ability (IC<SUB>50</SUB>: 16- and 4-fold dilution, respectively) and was inhibited dose-dependently by the FeSO₄-induced lipid peroxidation group (8- and 4-fold dilution: 2.3 and 1.5 nM, respectively). In MTT and LDH assays, 8-, 16-, 32- and 64-fold diluted HLJG0701 significantly increased cell viability by 70, 53, 35, and 26%, respectively. LDH released by HLJG0701 was reduced 1.3-fold with 8-fold diluted HLJG0701 compared to the H₂O₂-treated control. In addition, the inhibitory effect of HLJG0701 on oxidative stress in PC12 cells was confirmed by DCF-DA analysis (16-, 4-fold diluted HLJG0701: 50 and 68% ROS inhibition, respectively), TBARS (16- and 4-fold diluted HLJG0701: 50.7 and 46.5% inhibition, respectively), GPx (16- and 4-fold diluted HLJG0701: 133.3 and 227.3% release, respectively), and SOD analysis (16- and 4-fold diluted HLJG0701: 118.2 and 218.2% release, respectively). These results suggested that HLJG0701 protects neuronal cells by its anti-oxidative effects and hence can be a potential preventive material against neurodegenerative diseases.