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LED 보안등의 광균일도 향상을 위한 비구면 Mirror형 광학 설계에 관한 연구
정인호(In-Ho Jung),윤철용(Cheol-Yong Yoon),예인수(In-Soo Ye),현동훈(Dong-Hoon Hyun) 한국생산제조학회 2011 한국생산제조학회지 Vol.20 No.1
There is a limit to technology of improving luminous intensity uniformity and glare ,known as a weakness of existing LED lamp. Because of Using many LED, LED lamp not only waste energy but have bad effect on efficiency. Our goal is to develop security lights solution which is contented with suitable area in KS(Korean Industrial Standards) and to remove glare by combining asphere in optical system with different concept. To improve luminous intensity uniformity, a reflect mirror system was used after an aspheric optical system design for this study. We made a mirror and measured it after analysing luminance changes depend on aspheric shapes with simulation program to see if aspheric shapes effect luminance distribution. We made progress to find problems and improve them by comparing measured data and analysed data. This result of the study will contribute to industry of LED lighting through developing solution of emotional illumination of LED security lights by knowing the importance of reflectivity with this study and improving luminous intensity uniformity with solving the problem.
Cis-platin 내성 방광암세포주에 대한 Trichostatin A의 항암효과 분석
박지현(Jihyun Park),변석수(Seok-Soo Byun),이정은(Jeong Eun Lee),오종진(Jong Jin Oh),이상철(Sang Chul Lee),홍성규(Sung Kyu Hong),윤철용(Cheol Yong Yoon),이은식(Eunsik Lee),이상은(Sang Eun Lee) 대한비뇨기종양학회 2011 대한비뇨기종양학회지 Vol.9 No.2
Purpose: To determine anti-tumor effect of a histone deacetylase (HDAC) inhibitor, trichostatin A (TSA) in cis-platin resistant human bladder cancer cells (T24R2). Materials and Methods: T24 bladder cancer cell line and T24R2 were exposed to escalating dose of TSA and tumor cell proliferation was examined by CCK-8 assay. To evaluate the changes in cell cycle and apoptosis, flow cytometry was used and clonogenic assay was performed. Expression of p21WAF1/CIP1, cIAP1, XIAP, Bcl-2, and Bax were analyzed by Western blot. Results: Acute TSA administration (0.1-2μM for 24-72hours) suppressed tumor cell proliferation in a time- and dose-dependent manner (p<0.05) in all two bladder tumor cell lines. TSA induced G1 phase cell cycle arrest in both bladder cell lines and higher-fraction of sub-G1 (apoptotic portion) with 0.5μM in T24R2. A significant decrease in colony number was seen with the TSA treatment in the two cell lines. Western blot analysis revealed up-regulated p21 and bax, and down-regulated bcl-2, cIAP1 and xIAP with the increasing concentrations of TSA in both cell lines. Conclusions: Our results suggest anti-tumor effect of TSA in inhibiting bladder cancer growths and its potential role as an agent for treating cisplatin-resistant bladder cancer.