차량 성능 및 안정성 향상을 위한 $H_{\infty}$ 요 모멘트 강인제어

안우성,박종현,Ahn, Woo-Sung,Park, Jong-Hyeon 대한기계학회 2000 大韓機械學會論文集A Vol.24 No.8

This paper proposes a new $H_{\infty}$ yaw moment control scheme using brake torque switching for improving vehicle performance and stability especially in high speed driving. In the scheme, one wheel is selected, depending on the vehicle states, at which a brake torque for control is applied. Steering angles are modeled as a disturbance to the system and the $H_{\infty}$ controller is designed to minimize the difference between the performance of the vehicle and that of the desired model. Its performance robustness as well as stability robustness to system parameter variations is assured through ${\mu}$-analysis. Various simulations with a nonlinear 8-DOF vehicle model show that proposed controller enhances the vehicle performance and stability under disturbances and parameter variations as well as under the normal driving condition.

[후기] 제4차 IAA 서울 심포지엄 : 21세기 새로운 도시와 주거

안우성 새건축사협의회 2005 건축과사회 Vol.1 No.-

회원작품: 롯데국제교육과

안우성,Ahn, Woo-Sung 대한건축사협회 2010 建築士 Vol.2010 No.6

[서울시 공공건축가] 서울시 공공건축가제도를 통한 자문사례

안우성 새건축사협의회 2014 건축과사회 Vol.26 No.-

빙 및 일반해역 운항을 고려한 아라온호 선수프레임의 피로수명 계산

안우성,이탁기,황미란 한국해양공학회 2018 韓國海洋工學會誌 Vol.32 No.6

Ice-going ships such as icebreakers, icebreaking tankers, and icebreaking LNG carriers are subjected to wave loads in open water and ice loads in ice-covered water. In terms of the ship’s structural design, the local ice load is important. The fatigue failure due to repeated ice loads is also important. ISO 19906 specifies the assessment of the fatigue limit for a polar offshore structures. In addition, Lloyd’s Register refers to fatigue damage based on ShipRight FDA ICE. In ShipRight FDA ICE, the fatigue damage indices due to wave and ice loads are simply presented as 0.5 for each load. It also states that the sum of the two fatigue damage indices should not exceed one. This study calculated and analyzed the fatigue damage index and fatigue life considering ARAON's voyage schedules and the assumed Antarctic voyage based on data measured during the Arctic voyage of ARAON in 2010.

회원작품: 홍아기연 오정공장

안우성,Ahn, Woo-Sung 대한건축사협회 2011 建築士 Vol.2011 No.7

회원작품 - 탄현동성당

안우성,Ahn, Woo-sung 대한건축사협회 2014 建築士 Vol.2014 No.8

Induction of Mesenchymal to Epithelial Transition of Circulating Mesenchymal Stem Cells by Conditioned Medium of Injured Cornea

안우성,홍현숙,Mingzi Zhang,정은경,손영숙 한국조직공학과 재생의학회 2013 조직공학과 재생의학 Vol.10 No.2

We previously reported that substance-P (SP) mobilized mesenchymal stem cells (MSCSP) from bone marrow and recruited them to the injured site, which then participated in wound healing process by incorporation into the corneal epithelium in a rabbit alkali burn model. In the present study, we investigated if the injured microenvironment could stimulate the recruited MSCSP to lose their mesenchymal phenotype and then acquire epithelial phenotype or not. Especially, we focused on the effects of secreted soluble tissue factors of wound microenvironment in the rat cornea wound model. Priming the circulating MSCSP with conditioned medium of the injured tissue at day 5 elicited morphological and molecular changes similar to those noted during mesenchymal to epithelial transition (MET),which was accompanied by the reduction of alpha-smooth muscle actin (α-SMA), a typical mesenchymal marker,and induction of E-cadherin, a typical epithelial marker. Furthermore, these primed MSCSP rearranged their actin cytoskeletal system from distinct actin stress fiber projection to the cell-to-cell-contact-enriched actin microfilament. The primed cells also had increased survival in keratinocyte growth medium (KGM). In conclusion, MSCSP could be differentiated in vitro to the epithelial cells under the injured microenvironment, which may also occur in vivo during tissue repair.

Identifying a Molecular and Cellular Phenotype of Mesenchymal Stem Cells mobilized from Substance P in the Peripheral Blood

안우성,손영숙,정은경,장정호,임지은 한국조직공학과 재생의학회 2015 조직공학과 재생의학 Vol.12 No.2

Here we compared MSCs isolated from bone marrow (BMSC) and peripheral blood at 2 days after intravenoussubstance P (SP) injection (MSCSP) to define specific roles of circulating MSCs in tissue repair process. Themass analysis of changed genes over and under log2 fold showed 97% of genes were identical in these two groups. We found MSCSP increased the molecular signatures related to homing and engraftment (tetraspanin 8, tetraspanin12), extracellular matrix (ECM) (collagen type IV α1, nidogen 2, laminin-α5, basement membrane components;collagen type XII α1, collagen XV α1, ECM components linking basement membrane to stroma), transmigration(ICAM-1, P-selectin, endothelin), motility (Rho GTPase activating protein 8, myosin heavy chain 10 and 11, troponinC type 1 and 2), transdifferentiation potential (keratin 19, CMTM 8), immunity (IL-1α, IL-33, IL-1R type 1),and angiogenesis (endothelin1, VEGF-C, IL-33, laminin-α5) according to functional classification of the 3 % ofchanged genes. Functional differences were also shown in MSCSP in regulation of viability of immune cells, JurkatT cells and U937 monocytes. In addition, α-smooth muscle actin (α-SMA) significantly increased in MSCSP comparedwith BMSC, demonstrating committed differentiation to myofibroblast participated in tissue repair. However,intrinsic differentiation potential to mesenchymal tissues, adipogenesis and osteogenesis, was not altered in MSCSP. From the molecular and cellular phenotyping of MSCSP, circulating MSCSP seems to be a distinct population mobilizedfrom heterogenous BMSC with well adapted for the participation in tissue repair process by enhancing homing, engraftment, migration, and angiogenesis with multipotent differentiation potential to either myofibroblasts or epithelial-like cells. Here we compared MSCs isolated from bone marrow (BMSC) and peripheral blood at 2 days after intravenoussubstance P (SP) injection (MSCSP) to define specific roles of circulating MSCs in tissue repair process. Themass analysis of changed genes over and under log2 fold showed 97% of genes were identical in these two groups. We found MSCSP increased the molecular signatures related to homing and engraftment (tetraspanin 8, tetraspanin12), extracellular matrix (ECM) (collagen type IV α1, nidogen 2, laminin-α5, basement membrane components;collagen type XII α1, collagen XV α1, ECM components linking basement membrane to stroma), transmigration(ICAM-1, P-selectin, endothelin), motility (Rho GTPase activating protein 8, myosin heavy chain 10 and 11, troponinC type 1 and 2), transdifferentiation potential (keratin 19, CMTM 8), immunity (IL-1α, IL-33, IL-1R type 1),and angiogenesis (endothelin1, VEGF-C, IL-33, laminin-α5) according to functional classification of the 3 % ofchanged genes. Functional differences were also shown in MSCSP in regulation of viability of immune cells, JurkatT cells and U937 monocytes. In addition, α-smooth muscle actin (α-SMA) significantly increased in MSCSP comparedwith BMSC, demonstrating committed differentiation to myofibroblast participated in tissue repair. However,intrinsic differentiation potential to mesenchymal tissues, adipogenesis and osteogenesis, was not altered in MSCSP. From the molecular and cellular phenotyping of MSCSP, circulating MSCSP seems to be a distinct population mobilizedfrom heterogenous BMSC with well adapted for the participation in tissue repair process by enhancing homing,engraftment, migration, and angiogenesis with multipotent differentiation potential to either myofibroblasts or epithelial-like cells.

[발주제도] 강요된 컨소시엄은 정당한가

안우성 새건축사협의회 2013 건축과사회 Vol.24 No.-