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RISS 인기검색어
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- 저자 : 박중채(Joong Chae Park) (검색결과 2 건)
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박중채,김정년,장철호,최명재,김홍용,김정숙,정철영,Park, Joong-Chae,Kim, Jeong-Nyun,Chang, Cheol-Ho,Chey, Myung-Jae,Kim, Hong-Young,Kim, Jeong-Suk,Chung, Churl-Young 대한소아소화기영양학회 1998 Pediatric gastroenterology, hepatology & nutrition Vol.1 No.1
미숙아에서 유문협착증은 매우 드문 질환으로 증상이 비전형적이고 한번의 복부 초음파 검사로 진단이 어렵다. 저자들은 구토와 수유 곤란이 발생한 미숙아에서 계속적인 복부 초음파 검사와 위장관 조영술로 유문협착증으로 진단하였고 수술 후 증세 호전되어 퇴원한 2례를 보고하는 바이다. The incidence of Hypertrophic pyloric stenosis (HPS) in premature infants is rare, the presentation is not typical, and the diagnosis delayed due to uncertain diagnostic criteria in abdominal ultrasonography (US). We report two premature infants with HPS diagnosed by US and upper gastrointestinal (UGI) contrast study. Patient 1. A premature female infant (birth weight 1950 gm at 34 week's gestation) with the onset of intermittent vomiting at 9 days of age was evaluated. US was normal at 13 days of life, however, abnormal at 41 days of life (pyloric muscle length 16.5 mm). Patient 2. A premature male infant (birth weight 1470 gm at 29 week's gestation) with the onset of intermittent vomiting at 10 days of age was evaluated. US showed pylorospasm at 11 days of life, however, findings compatible with HPS at 57 days of life (pyloric muscle thickness 11 mm). UGI contrast study at 48 days of life showed similar findings in both cases. Both patients had undergone pyloromyotomy. In conclusion, the diagnosis of HPS in premature infants requires careful follow-up by US and UGI contrast study.
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박중채(Joong Chae Park),김홍동(Heung Dong Kim),이숙환(Sook Hwan Kee) 대한소아신경학회 1999 대한소아신경학회지 Vol.6 No.2
저자들은 전형적인 임상 증상과 뇌파 소견을 보인 환아에서 유전학적으로 염색체 15q11-13의 결실이 확인된 Angelman 증후군 1례를 경험하였기에 문헌 고찰과 함께 보고하는 바이다 Angelman Syndrome is a rare genetic disorder characterized by developmental delay, speech impairment, ataxic gait, paroxysmal laughter, and seizures. The diagnosis is suspected in infants who have the characteristic clinical features and electroencephalographic(EEG) abnormalities, and is confirmed by the genetic identification of a maternally derived 19q11-13 deletion. We report a case of genetically confirmed Angelman Syndrome who had the characteristic clinical and EEG features.
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