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도시숲과 가로수가 대기 흐름과 기온에 미치는 영향에 관한 수치 연구
강건,최원식,김재진,Kang, Geon,Choi, Wonsik,Kim, Jae-Jin 대한원격탐사학회 2022 大韓遠隔探査學會誌 Vol.38 No.6
This study investigated the effects of the urban forest and street trees on flow and temperature distribution in the Daegu National Debt Redemption Movement Memorial Park. For this, we implemented tree-drag and tree-cooling parameterization schemes in a computational fluid dynamics (CFD) model and validated the simulated wind speeds, wind directions, and air temperatures against the measured ones. We used the wind speeds, wind directions, air temperatures predicted by the local data assimilation and prediction system (LDAPS) as the inflow boundary conditions. To investigate the flow and thermal characteristics in the presence of trees in the target area, we conducted numerical experiments in the absence and presence of trees. In the absence of trees, strong winds and monotonous flows were formed inside the park, because there were no obstacles inducing friction. The temperature was inversely proportional to the wind speed. In the presence of trees, the wind speeds(temperatures) were reduced by more than 40 (5)% inside the park with a high planting density due to the tree drag (cooling) effect, and those also affected the wind speeds and temperatures outside the park. Even near the roadside, the wind speeds and temperatures were generally reduced by the trees, but the wind speeds and air temperatures increased partly due to the change in the flow pattern caused by tree drag.
강건,이유란,주희경,박명수,김국성,최성아,전병화 대한약리학회 2015 The Korean Journal of Physiology & Pharmacology Vol.19 No.5
Histone deacetylase (HDAC) has been recognized as a potentially useful therapeutic target for cardiovascular disorders. However, the effect of the HDAC inhibitor, trichostatin A (TSA), on vasoreactivity and hypertension remains unknown. We performed aortic coarctation at the inter-renal level in rats in order to create a hypertensive rat model. Hypertension induced by abdominal aortic coarctation was significantly suppressed by chronic treatment with TSA (0.5 mg/kg/day for 7 days). Nicotinamide adenine dinucleotide phosphate-driven reactive oxygen species production was also reduced in the aortas of TSA-treated aortic coarctation rats. The vasoconstriction induced by angiotensin II (Ang II, 100 nM) was inhibited by TSA in both endothelium-intact and endothelium-denuded rat aortas, suggesting that TSA has mainly acted in vascular smooth muscle cells (VSMCs). In cultured rat aortic VSMCs, Ang II increased p66shc phosphorylation, which was inhibited by the Ang II receptor type I (AT1R) inhibitor, valsartan (10 μM), but not by the AT2R inhibitor, PD123319. TSA (1∼10 μM) inhibited Ang II-induced p66shc phosphorylation in VSMCs and in HEK293T cells expressing AT1R. Taken together, these results suggest that TSA treatment inhibited vasoconstriction and hypertension via inhibition of Ang II-induced phosphorylation of p66shc through AT1R.