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Jang, Leex2010,Woon,Jeon, Daex2010,Woo,Kim, Myoung,Jeon, Jux2010,Won,Polyakov, Alexander Y.,Ju, Jinx2010,Woo,Lee, Seungx2010,Jae,Baek, Jongx2010,Hyeob,Yang, Jinx2010,Kyu,Lee, Inx2010,H WILEY‐VCH Verlag 2012 Advanced functional materials Vol.22 No.13
<P><B>Abstract</B></P><P>Localized surface plasmon (LSP) effects due to Ag and Ag/SiO<SUB>2</SUB> nanoparticles (NPs) deposited on GaN/InGaN multiquantum well (MQW) light‐emitting diode (LED) structures are studied. The colloidal NPs are synthesized by a sol‐gel method and drop‐cased on the LED structures. The surface density of NPs its controlled by the concentration of the NP solution. Theoretical modeling is performed for the emission spectrum and the electric field distribution of LSP resonance for Ag/SiO<SUB>2</SUB> NPs. Enhanced photoluminescence (PL) efficiency is observed in the LED structures and the amount of PL enhancement increases with increasing the surface density of Ag and Ag/SiO<SUB>2</SUB> NPs. These effects are attributed to resonance coupling between the MQW and LSP in the NPs. It is also shown that the PL enhancement attainable with Ag NPs and Ag/SiO<SUB>2</SUB> NPs is comparable, but the latter displays a much higher stability with respect to long‐term storage and annealing due to a barrier for NP agglomeration, Ag oxidation, and impurity diffusion provided by the SiO<SUB>2</SUB> shell.</P>
Jung, Chang H.,Yang, Yoox2010,Soo,Kim, Junx2010,Seob,Shin, Jaex2010,Il,Jin, Yongx2010,Su,Shin, Jae Y.,Lee, Jong H.,Chung, Koo M.,Hwang, Jae S.,Oh, Jung M.,Shin, Yeonx2010,Kyun,Kweon, Daex2 Blackwell Publishing Ltd 2008 FEBS JOURNAL Vol.275 No.12
<P>Soluble <I>N</I>‐ethylmaleimide sensitive‐factor attachment receptor (SNARE) proteins have crucial roles in driving exocytic membrane fusion. Molecular recognition between vesicle‐associated (v)‐SNARE and target membrane (t)‐SNARE leads to the formation of a four‐helix bundle, which facilitates the merging of two apposing membranes. Synthetic peptides patterned after the SNARE motifs are predicted to block SNARE complex formation by competing with the parental SNAREs, inhibiting neuronal exocytosis. As an initial attempt to identify the peptide sequences that block SNARE assembly and membrane fusion, we created thirteen 17‐residue synthetic peptides derived from the SNARE motifs of v‐ and t‐SNAREs. The effects of these peptides on SNARE‐mediated membrane fusion were investigated using an <I>in vitro</I> lipid‐mixing assay, <I>in vivo</I> neurotransmitter release and SNARE complex formation assays in PC12 cells. Peptides derived from the N‐terminal region of SNARE motifs had significant inhibitory effects on neuroexocytosis, whereas middle‐ and C‐terminal‐mimicking peptides did not exhibit much inhibitory function. N‐terminal mimicking peptides blocked N‐terminal zippering of SNAREs, a rate‐limiting step in SNARE‐driven membrane fusion. Therefore, the results suggest that the N‐terminal regions of SNARE motifs are excellent targets for the development of drugs to block SNARE‐mediated membrane fusion and neurotransmitter release.</P>
Experimental Study of the Runaway Current in the J-TEXT Tokamak
Y. H. Luo,Z. Y. Chen,X. Q. Zhang,D. W. Huang,W. Jin,Y. H. Huang,Y. Tang,J. C. Li,R. H. Tong,W. Yan,G. Zhuang 한국물리학회 2014 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.64 No.3
Major plasma disruptions in tokamaks often generate runaway currents, which contain electronswith energies of several tens of megaelectron-volts (MeV). These currents can cause substantialdamage when control is lost and the current hits the limiters or the vessel wall. The interactionbetween the runaway electrons and the impurities inside the plasma results in soft X-ray emission,which can provide detailed information about the runaway generation process and the confinementof runaway electrons. A vertical soft X-ray array at the top of Joint Texas Experimental Tokamak(J-TEXT) was used to study the runaway beams resulting from major disruptions. Runawayelectron production and confinement of runaway current were observed by using soft X-ray images.
X. S. Luo,J. Li,Y. L. Jin,C. P. Hu,D. Jia,S. P. Zhan,Y. Yu,M. Hua,H. T. Duan 대한금속·재료학회 2020 METALS AND MATERIALS International Vol.26 No.8
Tribological properties of AlCoCrCuFeNi high-entropy alloy were studied after annealing at various temperatures. X-raydiffraction, scanning electron microscopy, energy dispersive spectroscopy, microhardness tester and pin-on-disc tribometeranalyses were performed to reveal the microstructure, composition, microhardness and tribological behavior variations. Withthe heat treatment temperature increasing, time taken for friction coefficient going through the rapidly dropping down andthen into the stable period increases, the white sheets structures and their size in dendrite area of the AlCoCrCuFeNi alloybecomes bigger, however BCC content declined dramatically. Then, the average friction coefficient of the AlCoCrCuFeNi/Si3N4 sliding friction pair increase from 0.037 to 0.115, and the pin wear loss increase from 3 to 11 μm.
Cho, Lisa Y.,Yang, Jae Jeong,Ko, Kwangx2010,Pil,Park, Boyoung,Shin, Aesun,Lim, Min Kyung,Oh, Jinx2010,Kyoung,Park, Sohee,Kim, Yoon Jun,Shin, Haix2010,Rim,Yoo, Keunx2010,Young,Park, Sue K. Wiley Subscription Services, Inc., A Wiley Company 2011 International journal of cancer: Journal internati Vol.128 No.1
<P><B>Abstract</B></P><P>A subadditive effect of hepatitis B virus (HBV) and hepatitis C virus (HCV) coinfection is possible because superinfection of one virus tends to inhibit infection of the other virus. However, studies have reported inconsistent findings, and two meta‐analyses of studies from various countries (1998) and China (2005) reported a supraadditive effect for hepatocellular carcinoma (HCC) risk. Thus, we reevaluate HBV/HCV monoinfection and coinfection. Of 411 reports, we included 59 studies that assessed the association between HBV/HCV monoinfection and coinfection for HCC risk. HCC risk because of high/detectable HBV DNA and HBeAg infection was higher than HBsAg infection, whereas anti‐HCV <I>vs</I> anti‐HCV/HCV RNA was not different. Geographically, HCC risk was significantly higher in nonendemic than in HBV or HCV endemic areas. Subadditive effect for HCC risk was presented in recently published studies, cohort studies and studies conducted in HBV/HCV nonendemic areas; an additive effect was presented in studies conducted in HBV endemic areas; a supraadditive effect was presented in previously published studies, case‐control studies and studies conducted in HCV endemic areas. Our results suggest HBV/HCV coinfection for HCC risk is not significantly greater than HBV/HCV monoinfection, and HCC risk due to HBV or HCV is higher in nonendemic than endemic areas. The <I>p‐heterogeneity</I> was significant for most analyses, except HBV(+)/HCV(+) and HBV biomarker analyses. Prevention strategies targeted toward HBV or HCV monoinfected patients are needed. In addition, tailored prevention to reduce infectivity such as HBV markers (HBeAg, HBV DNA) is needed.</P>
Kim, Yu J.,Kim, Jux2010,Young,Yoon, Jin Y.,Kyung, Sun Y.,Lee, Sang P.,Jeong, Sung H.,Moon, Chanil,Park, Jeongx2010,Woong Blackwell Publishing Ltd 2011 Basic & clinical pharmacology & toxicology Vol.109 No.1
<P><B>Abstract: </B> Cigarette smoking is the principal cause of chronic obstructive pulmonary disease (COPD), especially emphysema, which is characterized by alveolar wall destruction and airspace enlargement. Apoptosis of lung structural cells is involved in the pathogenesis of COPD. Xanthine derivatives (aminophylline or theophylline) have been used for the treatment of COPD as a bronchodilator. But the effects of xanthine derivatives on apoptosis of the lung structural cells remain poorly understood, even though it is known that theophylline protects against ultraviolet irradiation–induced cell death in corneal epithelial cells. This study was designed to determine whether aminophylline would protect against cigarette smoke extract (CSE)–induced apoptosis in lung fibroblasts. We demonstrated that aminophylline protected against apoptosis of MRC‐5 cells at a relatively lower therapeutic range (10 μg/ml), resulting in a significant increase in cell viability occurring at 20% concentration after 8‐hr exposure. Annexin staining decreased from 68 ± 4% of the control to 12 ± 2% of aminophylline (10 μg/ml) pre‐treatment after 20% CSE exposure for 12 hr (<I>p </I><<I> </I>0.05). Aminophylline decreased caspase 3 and 8 activities and nuclear condensation or fragmentation in MRC‐5 cells after exposure to 20% CSE for 12 hr compared with control and high levels of aminophylline (>50 μg/ml) pre‐treatment. These findings suggest that aminophylline protected apoptosis of MRC‐5 cells through the inactivation of caspases 3 and 8 and could be an effective agent to reduce cigarette smoking–induced lung structural cell apoptosis.</P>
Microwave Absorption and Magnetic Properties of Cobalt Ferrites/Carbon Nanotubes Nanocomposites
B. B. Zhang,P. F. Wang,J. C. Xu,Y. B. Han,H. X. Jin,D. F. Jin,X. L. Peng,B. Hong,J. Li,J. Gong,H. L. Ge,Z. W. Zhu,X. Q. Wang 성균관대학교(자연과학캠퍼스) 성균나노과학기술원 2015 NANO Vol.10 No.5
Owing to the unique microstructure and the excellent dielectric properties, carbon nanotubes(CNTs) were decorated with CoFe2O4 nanoparticles to synthesize the CoFe2O4/CNTs nanocomposites by the solvothermal method. The phase structure, morphology, magnetic properties and microwave absorption performance of the as-prepared CoFe2O4/CNTs were characterized and discussed by X-ray diffraction (XRD), thermal gravity analysis (TGA), transmission electron microscope (TEM), vibrating sample magnetometer (VSM) and vector network analyzer (VNA). All results indicated that the diameter of CoFe2O4 nanoparticles decorating on the surface of CNTs increased with the solvothermal temperature. CoFe2O4/CNTs prepared at 180℃, 200℃ and 220℃ exhibited superparamagnetism, while the other samples presented ferromagnetism at room temperature. And with the increasing solvothermal temperature, the saturation magnetization and coercivity increased up to 72 emu/g and 2000 Oe for the sample prepared at 260℃ (S-26). And the reflection loss of CoFe2O4/CNTs nanocomposites increased with the solvothermal temperature up to -15.7 dB for S-26 with the bandwidth of 2.5 GHz.
Lee, Beom H.,Kim, Joo H.,Lee, Sun Y.,Jin, Hye Y.,Kim, Kwix2010,Joo,Lee, Jinx2010,Joo,Park, Jungx2010,Young,Kim, Gux2010,Hwan,Choi, Jinx2010,Ho,Kim, Kyung M.,Yoo, Hanx2010,Wook Blackwell Publishing Ltd 2011 Liver International Vol.31 No.6
<P><B>Abstract</B></P><P><B>Introduction and aims: </B> Wide phenotypic and genotypic heterogeneities in Wilson's disease (WD) have been reported, hampering the study of their correlations. The goal of this study was to identify the factors related to these diversities.</P><P><B>Methods: </B> Clinical courses and molecular genetic characteristics were analysed in 237 unrelated Korean WD families. The average follow‐up period was 8.2 ± 5.8 years.</P><P><B>Results: </B> Presenting phenotypes were classified as H1 (12.2%), H2 (42.4%), N1 (21.6%), N2 (0.4%), NX (0.4%), presymptomatic (22.4%) and other (0.4%), modifying the guidelines by Ferenci and colleagues. Age at presentation was youngest and cirrhosis was rarest in the presymptomatic group. Decompensated cirrhosis was the highest in the H1 group. Favourable outcome was rarest in the N1 group. Forty‐seven (11 novel) <I>ATP7B</I> mutations were identified in 85% of the 474 alleles. Multiplex ligation‐dependent probe amplification assays in <I>ATP7B</I> and analyses of <I>ATOX1</I> and <I>COMMD1</I> genes identified no additional mutations. Yeast complementation assays demonstrated functional perturbation of the seven novel missense mutants. Five major mutations, p.Arg778Leu, p.Ala874Val, p.Asn1270Ser, p.Lys838SerfsX35 and p.Leu1083Phe, accounted for 63% of the alleles. H1 was more common, age at presentation was younger and N1+N2+NX tended to be less common in patients with nonsense, frame shifting or splicing mutations than in those with missense mutations alone. Patients with both mutations in the transduction (Td) or the ATP hinge domain showed presymptomatic or hepatic manifestations but no neurological manifestation.</P><P><B>Conclusions: </B> The presenting phenotype strongly affects the clinical outcome of WD, and is related to the <I>ATP7B</I> mutation type and location, providing an evidence for genotype–phenotype correlations in WD.</P>