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음식물류폐기물 공공 자원화시설 운영에 관한 연구 (Ⅱ)
홍용표ㆍ김웅용ㆍ신현곤(Yong Pyo HongㆍUng Yong KimㆍHyun Gon Shin) 유기성자원학회 2016 유기물자원화 Vol.24 No.1
본 연구는 음식물류폐기물 공공 자원화 시설 운영의 문제점과 제도 개선방안을 위하여 실시하였다. 현재 운영중인 음식물류 폐기물의 가동율 현황과 자원화 시설의 확충 계획에 대한 분석을 통해 시설의 무분별한 신규건설로 인한 국가의 예산이 낭비되지 않도록 그 대안을 제시하였으며, 그 결과는 다음과 같다. 공공 자원화시설 신규 확충보다는 기존 민간 자원화시설을 활용하여 예산을 절약하고 음식물류 폐기물의 안정적인 처리는 물론 자원순환을 촉진시킬 수 있는 정책전환이 필요하다. 음식물류 폐기물을 친환경적인 방법으로 안정적으로 처리하기 위해서는 제도개선이 필요하며, 이를 위해 민간 자원화시설 가동률 저하를 줄일 수 있는 준공영제 도입이 필요하다. 또한, 공공 자원화시설의 검증된 기술과 경제성 확보 방안 마련이 필요하다. This study is conducted to find out the problems of the public food waste recycling facility operation and its improvement. In order to save the national budget by analyzing plans to expand including operation rate for the food waste recycling facility, the alternative ways are suggested. The result can be as follows: Policy switch is required not only to save budget and stable disposal of food waste but also to promote resource recycling rather than expanding public facilities. In order to change the policy for stable and eco-friendly disposal of food waste, it is necessary to adopt semipublic system that prevent the decline of the rate of operation of private resource recycling facility. Furthermore, it is required to prepare both qualified technologies of public resource recycling and the ways of profitability secure.
재래 흑돼지와 중국 재래돈간의 Melanocortin Receptor 1(MC1R) 유전자의 유전자형 분석
이성수,양보석,정진관,고서봉,오성종,양영훈,김규일,이찬동,풍서당 한국동물자원과학회 2001 한국축산학회지 Vol.43 No.1
PCR-RFLP analysis of Melanocortin receptor 1 (MC1R) gene was carried out to investigate the genotype distribution in Korean-Native and Chinese-Native pig breeds(Neijiang, Putian, Wannanhua and Jinhua). Allelic variants of MC1R in pigs were analyzed by digestion of BspH I , AccII and Hha I . Ncijiang. Wannanhua and Jinhua had only MC1R*2 allele which is considered to be typical genotype for Meishan and Large Black. Among 20 Korean-Native Pigs and 5 Putian pigs, 10 and 2 heads had the MC1R*3 allele (*2/3 or 3/3), respectively, which was detected in European pig breeds such as Hampshire, Large White, and Pietrain. The remaining animals possessed the genotype MC1R*2/2, probably reflecting the genetic introgression of MC1R*3 allele into Korean-Native and Putian pig breeds by the crossbreeding for improvement. The alleles MC1R*l and MC1R*4, which are considered to be typical allele for European Wild Boar with wild-type coat color and Duroc with red coat color, respectively, were not detected in pig breeds used in this experiment. These results indicate that the analysis of genotype frequencies of MC1R gene may be a useful tool for the conservation of Korean-Native and Chinese-Native pig breeds.
김현진(HJ Kim),김옥준(OJ Kim),김대윤(DY Kim),정지연(JY Jung),김민석(MS Kim),김원재(WJ Kim),최홍란(HR Choi),이은주(EJ Lee),김선헌(SH Kim) 대한해부학회 2003 Anatomy & Cell Biology Vol.36 No.2
Ipriflavone (isopropoxyisoflavone)은 soy isoflavone daidzein으로부터 합성된 isoflavone 일종으로 osteoporosis나 osteopenia 등에 이용되고 있다. 그러나 ipriflavone이 일차적으로 뼈 흡수를 억제함으로써 뼈 질환 치료에 임상적 효과를 갖는지 또는 뼈 형성을 촉진하는지에 대하여는 확실하지 않다. 또한 임상적 효능에 대한 효과가 대부분 골다공증의 치료 등에 국한되어 있는 반면, 단백 수준의 뼈 형성 인자의 유도 효과나 정상 뼈의 재생 효과를 밝히는 연구는 드물다. 본 연구는 in vitro에서 ipriflavone에 의한 뼈형성인자 발현에 대한 영향을 구명하고, 이를 생체에 국소적으로 직접 적용함으로써 ipriflavone의 뼈 재생 효과를 구명하고자 시도되었다. 시험관내 실험 : Ipriflavone 처리 3일 후 MC3T3-E1 뼈모세포주의 세포증식 효과는 10-8 M에서 10-4 M까지 대조군과 유 의한 차이가 없었다. 뼈 형성 지표로 알려진 collagen Iα1(I) 유전자 발현은 10-8 M에서 10-5 M까지 모든 농도에서 증가하였고 10-6 M에서 가장 많이 발현되었다. 그러나 10-4 M과 같이 높은 농도에서는 다시 크게 감소하여 대조군과 큰 차이를 보이지 않았다. alkaline phosphatase 발현은 대조군과 vehicle군과 같이 낮은 농도에서 큰 증가를 보이지 않았으나 10-5 M 농도에서 크게 증가하였다. 생체 실험 : Ipriflavone 투여 3일군에서 뼈 결손부는 대부분 혈병으로 채워져 있었고 혈병내에는 많은 혈액세포가 산재 하였으며, 실험군에서 대조군에 비하여 섬유원이 풍부하게 존재하였다. 약제 투여 7일 후 대조군과 실험군에서 모두 뼈 결손부 중심은 부분적으로 신생뼈에 의하여 재생되는 소견을 보였으며 대조군의 신생뼈는 풋뼈조직으로 구성되어 있었으 나, 실험군의 신생뼈는 일차해면뼈 조직으로 구성되어 있었다. 약제 투여 14일군에서 뼈 결손부가 뼈잔기둥 형태의 신생 뼈에 의하여 대치되어 있었으며 대조군에 비하여 실험군에서 더 많은 뼈조직으로 재생되어 있었다. 상기 연구 결과는 ipriflavone이 적정 농도에서 뼈 형성에 기여하며 뼈 결손부 재생 치유에 유용하게 사용될 수 있음을 시사한다. Ipriflavone (isopropoxyisoflavone) is a synthetic derivative from soy isoflavone daidzein. There are compelling evidences that it has an inhibitory effect on bone resorption in vitro and in vivo. It is not clarified however, whether its anti-osteopenic effect derives from inhibition of bone resorption or stimulation of bone formation. The present study aims to elucidate effects of ipriflavone on osteoblasts gene expression and possible clinical application on bone regeneration. Three days’ continuous treatment of ipriflavone had no effects on MC3T3-E1 osteoblastic cell proliferation at 10-8M to 10-4 M concentration from MTS assay. Gene expression of collagen α1(I), which is a bone formation marker, was markedly increased by ipriflavone treatment at 10-9 M to 10-5 M, but decreased to control level at 10-6 M concentration. Alkaline phosphatase, another bone formation marker, was also increased at its gene expression at 10-5 M concentration. Artificial bone defects on the rat calvaria were healed with hematoma and much more profuse and delicate fibrin at 3 days treatment of ipriflavone. Primary spongy bone was noted in bony defect center after 7 days treatment of ipriflavone, in contrast to woven bone in control group. Newly formed trabecular bone could be attained by the treatment after 14 days. These results suggested that ipriflavone can affect on osteoblasts at molecular level, and be clinically applied for local bone regeneration.
Incidence and Clinical Characteristics of Congenital Cardiac Anomaly in Korea
위지선 ( Ji Sun We ),( Bo Hyun Yoon ),( Ahm Kim ),( Jong Hwa Kim ),( Yong Kyon Jo ),( Yong Ju Kim ),( Moon Young Kim ),( Suk Young Kim ),( Sung Soo Kim ),( Young Woo Kim ),( Byung In Kim ),( Jong Chul Sh 대한산부인과학회 2008 대한산부인과학회 학술대회 Vol.94 No.-
Kim, SH.,Choi, JH.,Lee, KW.,Kim, SH.,Shin, ES.,Oh, HB.,Suh, CH.,Nahm, DH.,Park, HS. Blackwell Scientific Publications 2005 Clinical and experimental allergy Vol.35 No.3
<P>Summary</P><P>Background</P><P>Urticaria/angioedema is a common aspirin-induced allergy; however, its pathogenic mechanism is not understood.</P><P>Objective</P><P>In order to uncover the genetic mechanism, we studied the associations of the human leucocyte antigen (HLA) genotypes in patients with aspirin-induced urticaria compared with aspirin-intolerant asthma and normal control in a Korean population.</P><P>Methods</P><P>Ninety-four aspirin-induced urticaria patients presenting urticaria/angioedema-induced by both ASA and NSAID (50 had underlying chronic urticaria) and showing positive responses on oral aspirin challenge test, 76 aspirin-intolerant asthmatics with positive responses on lysine–aspirin bronchoprovocation test, and 185 normal healthy controls were enrolled. HLA-DRB1, DQB1, and DPB1 genotypings were performed by direct DNA sequencing analysis.</P><P>Results</P><P>The allele frequencies of HLA-DRB1<SUP>*</SUP>1302 (18.1%) and HLA-DQB1<SUP>*</SUP>0609 (10.1%) in aspirin-induced urticaria were significantly higher than in aspirin-intolerant asthma (5.3%, <I>P</I>=0.0004; 2.0%, <I>P</I>=0.0024) and in normal controls (8.1%, <I>P</I>=0.0005; 3.2%, <I>P=</I>0.0008), and they remained significant after correcting for multiple comparisons. The patients with these two HLA markers had a significantly younger age than patients without, while no associations were found in with respect to atopic status, a history of previous allergic diseases, total IgE level, or presence of underlying chronic urticaria (<I>P</I>>0.05, respectively). In haplotype analysis, the HLA-DRB1<SUP>*</SUP>1302-DQB1<SUP>*</SUP>0609-DPB1<SUP>*</SUP>0201 was significantly higher in the aspirin-induced urticaria (8.0%) than in the aspirin-intolerant asthma (0.7%, <I>P</I>=0.0014) and normal controls (2.0%, <I>P</I>=0.0006).</P><P>Conclusion</P><P>These findings suggest that the HLA-DRB1<SUP>*</SUP>1302-DQB1<SUP>*</SUP>0609-DPB1<SUP>*</SUP>0201 may be a strong genetic marker to determine the aspirin-induced urticaria phenotype.</P>