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Hydrothermal deactivation over CuFe/BEA for NH3-SCR
Qingjin Lin,Xi Feng,Hailong Zhang,Chenlu Lin,Shuang Liu,Haidi Xu,Yaoqiang Chen 한국공업화학회 2018 Journal of Industrial and Engineering Chemistry Vol.65 No.-
Hydrothermal deactivation over CuFe/BEA and CuFe/BEA-HT for the selective catalytic reduction of NO with NH3 (NH3-SCR) were investigated with different characterizations. The hydrothermal treatment destroys BEA skeleton, losing acid sites reacting with the aerial NOx. H2-TPR and XPS reveal that the hydrothermal treatment could weaken the interaction between Cu/Fe species and the surrounding other atoms, causing the aggregation of isolated and low nuclearity copper/iron species. Aggregated copper/iron oxides depress the redox property of CuFe/BEA-HT, producing the lower concentration of NO2. Besides, results of in situ DRIFTS also indicate that both CuFe/BEA and CuFe/BEA-HT follow Eley–Rideal (E–R) mechanism at 225 °C.
( Xiaofeng Zhou ),( Jianghui Li ),( Weilong Wang ),( Fan Yang ),( Bingqian Fan ),( Chenlu Zhang ),( Xiaojun Ren ),( Feng Liang ),( Rong Cheng ),( Fengying Jiang ),( Huaibin Zhou ),( Juanjuan Yang ),( 한국미생물 · 생명공학회 2020 Journal of microbiology and biotechnology Vol.30 No.7
Various genetically engineered microorganisms have been developed for the removal of heavy metal contaminants. Metal biosorption by whole-cell biosorbents can be enhanced by overproduction of metal-binding proteins/peptides in the cytoplasm or on the cell surface. However, few studies have compared the biosorption capacity of whole cells expressing intracellular or surface-displayed metal-adsorbing proteins. In this study, several constructs were prepared for expressing intracellular and surface-displayed Ochrobactrum tritici 5bvl1 ChrB in Escherichia coli BL21(DE3) cells. E. coli cells expressing surface-displayed ChrB removed more Cr(VI) from aqueous solutions than cells with cytoplasmic ChrB under the same conditions. However, intracellular ChrB was less susceptible to variation in extracellular conditions (pH and ionic strength), and more effectively removed Cr(VI) from industrial wastewater than the surface-displayed ChrB at low pH (<3). An adsorptiondesorption experiment demonstrated that compared with intracellular accumulation, cell-surface adsorption is reversible, which allows easy desorption of the adsorbed metal ions and regeneration of the bioadsorbent. In addition, an intrinsic ChrB protein fluorescence assay suggested that pH and salinity may influence the Cr(VI) adsorption capacity of ChrB-expressing E. coli cells by modulating the ChrB protein conformation. Although the characteristics of ChrB may not be universal for all metal-binding proteins, our study provides new insights into different engineering strategies for whole-cell biosorbents for removing heavy metals from industrial effluents.
Genome Architecture and Its Roles in Human Copy Number Variation
Chen, Lu,Zhou, Weichen,Zhang, Ling,Zhang, Feng Korea Genome Organization 2014 Genomics & informatics Vol.12 No.4
Besides single-nucleotide variants in the human genome, large-scale genomic variants, such as copy number variations (CNVs), are being increasingly discovered as a genetic source of human diversity and the pathogenic factors of diseases. Recent experimental findings have shed light on the links between different genome architectures and CNV mutagenesis. In this review, we summarize various genomic features and discuss their contributions to CNV formation. Genomic repeats, including both low-copy and high-copy repeats, play important roles in CNV instability, which was initially known as DNA recombination events. Furthermore, it has been found that human genomic repeats can also induce DNA replication errors and consequently result in CNV mutations. Some recent studies showed that DNA replication timing, which reflects the high-order information of genomic organization, is involved in human CNV mutations. Our review highlights that genome architecture, from DNA sequence to high-order genomic organization, is an important molecular factor in CNV mutagenesis and human genomic instability.
Chen Lu,Cao Kai,Gu Yurong,Luo Chao,Mao Wei,Zhou Weijun,Zhu Jinwei,Zhang Huying 대한독성 유전단백체 학회 2021 Molecular & cellular toxicology Vol.17 No.3
Background Kaempferol (KMF) is a fl avone reported to have anti-oxidant and anti-infl ammatory activity. Objective The present study screened the eff ect of KMF in the animal model of spinal cord injury (SCI). Results KMF caused a signifi cant inhibition of spinal cord injury mediated oxidative stress and also suppressed the infl ammatory reactions. The treatment of KMF also inhibited the levels of p53, TGF-β1 and COX-2 whereas a signifi cant elevation in Bcl-2/Bax ratio was observed after the rats were treated with KMF. The in silico docking analysis suggested potential binding of KMF having lower energy with p53 confi rming the potential target of KMF. Conclusion The treatment of KMF exerted neuroprotective eff ect by improving the anti-oxidant status and infl ammatory response. The signifi cant spinal cord injury protective eff ect of KMF in rats was attributed by targeting p53 and Bcl-2/Bax ratio.