http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
( Bong Gyu Kwak ),( Kyun Young Kim ),( Suyun Oh ),( Yu Ah Hong ),( Hyeon Seok Hwang ),( Yoon Kyung Chang ),( Suk Young Kim ) 대한내과학회 2016 대한내과학회 추계학술발표논문집 Vol.2016 No.1
Posterior reversible encephalopathy syndrome (PRES) is clinical and radiologic syndrome that accompanied by a headache, altered mental state, seizures and other neurological signs with radiologically by reversible changes on brain image. Although the pathophysiology of PRES is incompletely understood, renal failure was known as one of the risk factors. Regarding end-stage renal disease (ESRD) and PRES, only a few cases of adults on hemodialysis have been described in the literature. We report an interesting case of PRES with severe hypertension receiving chronic hemodialysis. A 55-year-old male with ESRD was admitted to our hospital due to traumatic hemothorax and performed video-assisted thoracoscopic surgery with chest tube insertion. During 3 weeks after operation, his systolic blood pressure (BP) was very huge fluctuation between 80 mmHg to 200 mmHg. Despite using anti-hypertensive medication and hemodialysis, it is very difficult to control BP and pulmonary congestion. Suddenly he was observed decreased mentality and had a seizure-like movement with pulseless electrical activity. He was returned to spontaneous rhythm after successful cardiopulmonary resuscitation for 4 minutes with comatose mentality. We found symmetrical white matter edema in posterior cerebrum in T2-wighted Brain MRI and administrated anticonvulsant and performed continuous renal replacement therapy (CRRT) for 23 days to maintain systolic BP between 100 mmHg to 120 mmHg. His neurologic symptoms had disappeared completely after we controlled strict blood pressure. Hemodialysis patients presenting with severe hypertension and subsequent seizures and unconsciousness should undergo brain MRI to rule out the possibility of PRES and consider to maintain appropriate BP and volume control using CRRT.
Bong Gyu Kim,Han Bok Kwak,Eun-Yong Choi,Hun Soo Kim,Myung Hee Kim,Seong Hwan Kim,Min-Kyu Choi,Churl Hong Chun,Jaemin Oh,Jeong-Joong Kim 대한해부학회 2010 Anatomy & Cell Biology Vol.43 No.4
Among the several rotenoids, amorphigenin is isolated from the leaves of Amopha Fruticosa and it is known that has anti-proliferative effects and anti-cnacer effects in many cell types. The main aim of this study was to investigate the effects of amorphigenin on osteoclast differentiation in vitro and on LPS treated inflammatory bone loss model in vivo. We show here that amorphigenin inhibited RANKL-induced osteoclast differentiation from bone marrow macrophages in a dose dependent manner without cellular toxicity. Anti-osteoclastogenic properties of amorphigenin were based on a down-regulation of c-fos and NFATc1. Amorphigenin markedly inhibited RANKL-induced p38 and NF-κB pathways, but other pathways were not affected. Micro-CT analysis of the femurs showed that amorphigenin protected the LPS-induced bone loss. We concluded that amorphigenin can prevent inflammation-induced bone loss. Thus we expect that amorphigenin could be a treatment option for bone erosion caused by inflammation.
Z-scan 방법에 의한 비정질 As₂S₃ 박막의 비선형 굴절률 측정
김성규(Seong Gyu Kim),이영락(Yeung Lak Lee),곽종훈(Chong Hoon Kwak),최옥식(Ok Shik Choe),이윤우(Yun Woo Lee),송재봉(Jae Bong Song),서호형(Ho Hyung Suh),이일항(El-Hang Lee) 한국광학회 1998 한국광학회지 Vol.9 No.5
비선형 Kerr 매질을 통과하는 Gaussian 빔에 대해 aberration-free approximation과 Huygens-Fresnel 회절 이론을 적용하여 Z-scan 투과율에 대한 해석해를 유도하였다. 비정질 As₂S₃ 박막에 대해 Z-scan 실험을 수행하였으며 이론과 비교하여 잘 일치함을 알았다. 633㎚ 파장에서 측정된 비선형 굴절률 γ의 크기와 기호는 +8.65×10^(-6)㎠/W이며, 또한 먼 영역(far-field)에서 빔 세기분포를 측정하여 자기 집광(self-focusing) 효과를 가시적으로 확인하였다. We present a theoretical analysis of Gaussian beam propagation in nonlinear Kerr media by using aberration-free approximation and Huygens-Fresnel diffraction integral and obtain a simple analytic formular for Z-scan characteristics. Z-scan experiments are carried out on amorphous As₂S₃ thin film and compared with the theory developed, showing good agreement. The sign and the value of γ have been measured at 633 ㎚ to be +8.65×10^(-6)㎠/W. We also measured the far-field intensity profiles, which confirm again self-focusing effect.
Lee, Ji-Young,Kim, Bong Jo,Sim, Gyujin,Kim, Gyu-Tae,Kang, Dawon,Jung, Jae Hun,Hwa, Jeong Seok,Kwak, Yeon Ju,Choi, Yeon Jin,Park, Young Sook,Han, Jaehee,Lee, Cheol Soon,Kang, Kee Ryeon The Korean Academy of Medical Sciences 2011 JOURNAL OF KOREAN MEDICAL SCIENCE Vol.26 No.6
<P>The influence of spinal cord injury (SCI) on protein expression in the rat urinary bladder was assessed by proteomic analysis at different time intervals post-injury. After contusion SCI between T9 and T10, bladder tissues were processed by 2-DE and MALDI-TOF/MS at 6 hr to 28 days after SCI to identify proteins involved in the healing process of SCI-induced neurogenic bladder. Approximately 1,000 spots from the bladder of SCI and sham groups were visualized and identified. At one day after SCI, the expression levels of three protein were increased, and seven spots were down-regulated, including heat shock protein 27 (Hsp27) and heat shock protein 20 (Hsp20). Fifteen spots such as S100-A11 were differentially expressed seven days post-injury, and seven proteins including transgelin had altered expression patterns 28 days after injury. Of the proteins with altered expression levels, transgelin, S100-A11, Hsp27 and Hsp20 were continuously and variably expressed throughout the entire post-SCI recovery of the bladder. The identified proteins at each time point belong to eight functional categories. The altered expression patterns identified by 2-DE of transgelin and S100-A11 were verified by Western blot. Transgelin and protein S100-A11 may be candidates for protein biomarkers in the bladder healing process after SCI.</P>
S-490 sarcoidosis presenting as pancreatitis, duodenal ulcer and severe acute kidney injury
( Kyun Young Kim ),( Bong Gyu Kwak ),( Suyun Oh ),( Won Seok Park ),( Yoon Kyung Chang ),( Suk Young Kim ),( Hyeon Seok Hwang ),( Yu Ah Hong ) 대한내과학회 2016 대한내과학회 추계학술발표논문집 Vol.2016 No.1
Sarcoidosis is a multisystemic inflammatory disease characterized by the presence of noncaseating granulomas, mainly the lung and lymph nodes. We report an interesting case of sarcoidosis simultaneously involving the gastrointestinal tract, pancreas, and kidney and presenting as severe acute kidney injury (AKI). A 41-year-old man was admitted to our hospital with anorexia and weight loss over several months. Four months ago, he was diagnosed with idiopathic acute pancreatitis and recovered after steroid treatment. At that time, abdominal computed tomography (CT) showed diffuse pancreas swelling without solid mass. We started hemodialysis due to severe AKI with hypercalcemia and renal biopsy was performed. The histopathology indicated granulomatous interstitial nephritis with diffuse interstitial inflammation and noncaseating granulomas. Esophagogastroduodenoscopy revealed ulcerative and edematous mucosa on the stomach and the second part of the duodenum. Chest CT showed a slight interstitial pattern but definite hilar lymphadenopathy was not observed. Noncaseating granulomas were observed on microscopic examination in stomach, duodenum and bronchus. There was no involvement of sarcoidosis in any other organs. He received pulse methylprednisolone followed by maintenance prednisone therapy. After 2 months of treatment, renal function and hypercalcemia were improved. We suggest that renal sarcoidosis can manifest without typical pulmonary involvement and unusual extrathoracic organ involvement occurs with renal sarcoidosis presenting as severe AKI. Active enforcement of biopsy for suspicious lesions including kidney would provide the diagnostic clue in sarcoidosis with unusual manifestation and it supports the histological evidence of granulomas consistent with sarcoidosis. Keywords: sarcoidosis; kidney; pancreas; duodenum